贲门失弛缓症中氮能舒张功能受损的新机制。
Novel mechanism for impaired nitrergic relaxation in achalasia.
作者信息
Boeckxstaens G E
机构信息
Department of Gastroenterology and Hepatology, Academic Medical Centre, Meibergdreef 9, 1105AZ Amsterdam, the Netherlands.
出版信息
Gut. 2006 Mar;55(3):304-5. doi: 10.1136/gut.2005.078402.
Abstract
New insights into the pathogenesis of achalasia indicate that incubation with serum from patients with achalasia leads to altered neurochemical coding of the myenteric plexus and impairs the nitrergic response to nerve stimulation
摘要
贲门失弛缓症发病机制的新见解表明，用贲门失弛缓症患者的血清孵育会导致肌间神经丛神经化学编码改变，并损害对神经刺激的一氧化氮能反应
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本文引用的文献
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2
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