Leng Lin, Bucala Richard
Department of Medicine, Yale University School of Medicine, The Anlyan Center, S525, 300 Cedar Street, New Haven CT 06520-8056, USA.
Cell Res. 2006 Feb;16(2):162-8. doi: 10.1038/sj.cr.7310022.
The recent cloning of MIF receptor fills an important gap in our understanding of the molecular biology and immunology of MIF. The MIF receptor, like MIF, does not fall into any established family of protein mediators, providing both new challenges and opportunities for the structural and functional analysis of MIF signal transduction.
最近对巨噬细胞移动抑制因子（MIF）受体的克隆填补了我们在MIF分子生物学和免疫学认识上的一个重要空白。与MIF一样，MIF受体不属于任何已确立的蛋白质介质家族，这为MIF信号转导的结构和功能分析带来了新的挑战和机遇。
