Roos-Jansåker Ann-Marie, Renvert Helena, Lindahl Christel, Renvert Stefan
Department of Health Sciences, Kristianstad University, Sweden.
J Clin Periodontol. 2006 Apr;33(4):296-301. doi: 10.1111/j.1600-051X.2006.00908.x.
The aim of the present paper was to analyse, on patient and implant basis, factors related to peri-implant lesions.
Two hundred and eighteen patients treated with titanium implants were examined for biological complications at existing implants 9-14 years after initial therapy. The effects of several potentially explanatory variables, both on patient and on implant levels, were analysed.
On the implant level, the presence of keratinized mucosa (p = 0.02) and plaque (p = 0.005) was associated with mucositis (probing depth > or =4 mm + bleeding on probing). The bone level at implants was associated with the presence of keratinized mucosa (p = 0.03) and the presence of pus (p < 0.001). On the patient level, smoking was associated with mucositis, bone level and peri-implantitis (p = 0.02, <0.001 and 0.002, respectively). Peri-implantitis was related to a previous history of periodontitis (p = 0.05).
Individuals with a history of periodontitis and individuals who smoke are more likely to develop peri-implant lesions.
本文旨在基于患者和种植体分析与种植体周围病变相关的因素。
对218例接受钛种植体治疗的患者在初始治疗9至14年后对现有种植体的生物并发症进行检查。分析了几个潜在解释变量在患者和种植体水平上的影响。
在种植体水平上,角化黏膜的存在(p = 0.02)和菌斑(p = 0.005)与黏膜炎(探诊深度≥4 mm + 探诊出血)相关。种植体处的骨水平与角化黏膜的存在(p = 0.03)和脓液的存在(p < 0.001)相关。在患者水平上,吸烟与黏膜炎、骨水平和种植体周围炎相关(分别为p = 0.02、<0.001和0.002)。种植体周围炎与牙周炎既往史相关(p = 0.05)。
有牙周炎病史的个体和吸烟者更易发生种植体周围病变。