缺氧发作诱发大鼠海马切片CA1区锥体细胞兴奋性增高：GABAA和GABAB受体的作用 - Suppr

Suppr

超能文献

Hyperexcitability of pyramidal neurons in field CA1 in hippocampal slices from rats evoked by episodes of hypoxia: the roles of GABAA and GABAB receptors.

作者信息

Levin S G, Godukhin O V

机构信息

Institute of Theoretical and Experimental Biophysics, Russian Academy of Sciences, Pushchino, Moscow Region.

出版信息

Neurosci Behav Physiol. 2006 May;36(4):415-8. doi: 10.1007/s11055-006-0033-9.

DOI:10.1007/s11055-006-0033-9

PMID:16583170

Abstract

摘要

相似文献

Hyperexcitability of pyramidal neurons in field CA1 in hippocampal slices from rats evoked by episodes of hypoxia: the roles of GABAA and GABAB receptors.缺氧发作诱发大鼠海马切片CA1区锥体细胞兴奋性增高：GABAA和GABAB受体的作用

Neurosci Behav Physiol. 2006 May;36(4):415-8. doi: 10.1007/s11055-006-0033-9.

Changes in the excitability of pyramidal neurons in hippocampal slices from aged rats in conditions of periodic transient episodes of hypoxia.老龄大鼠海马脑片在周期性短暂缺氧发作条件下锥体细胞兴奋性的变化

Neurosci Behav Physiol. 2005 Nov;35(9):937-41. doi: 10.1007/s11055-005-0149-3.

[Hyperexcitability of CA1 pyramidal neurons induced by repeated brief episodes of hypoxia in the hippocampal slices: involment of GABAA and GABAB receptors].[海马切片中反复短暂缺氧诱导CA1锥体神经元的过度兴奋性：GABAA和GABAB受体的参与]

Ross Fiziol Zh Im I M Sechenova. 2005 May;91(5):581-6.

Enhancement of the synchronized firing of CA1 pyramidal cells by medial septum preconditioning: time-dependent involvement of muscarinic cholinoceptors and GABAB receptors.内侧隔区预处理增强CA1锥体细胞的同步放电：毒蕈碱型胆碱能受体和GABAB受体的时间依赖性参与

Neurosci Lett. 2006 Jan 23;393(1):1-6. doi: 10.1016/j.neulet.2005.09.035. Epub 2005 Oct 19.

GABAB receptor- and metabotropic glutamate receptor-dependent cooperative long-term potentiation of rat hippocampal GABAA synaptic transmission.大鼠海马GABAA突触传递的GABAB受体和代谢型谷氨酸受体依赖性协同长时程增强

J Physiol. 2003 Nov 15;553(Pt 1):155-67. doi: 10.1113/jphysiol.2003.049015. Epub 2003 Sep 8.

Neuroprotective effects of interleukin-10 and tumor necrosis factor-alpha against hypoxia-induced hyperexcitability in hippocampal slice neurons.白细胞介素-10和肿瘤坏死因子-α对海马切片神经元缺氧诱导的过度兴奋的神经保护作用。

Neurosci Lett. 2007 Apr 18;416(3):236-40. doi: 10.1016/j.neulet.2006.12.059. Epub 2007 Feb 24.

Do GABAA and GABAB inhibitory postsynaptic responses originate from distinct interneurons in the hippocampus?γ-氨基丁酸A型（GABAA）和γ-氨基丁酸B型（GABAB）抑制性突触后反应是否源自海马体中不同的中间神经元？

Can J Physiol Pharmacol. 1997 May;75(5):520-5.

The GABAA receptor-mediated recurrent inhibition in ventral compared with dorsal CA1 hippocampal region is weaker, decays faster and lasts less.与背侧CA1海马区相比，腹侧CA1海马区中GABAA受体介导的反复抑制作用较弱，衰减更快且持续时间更短。

Exp Brain Res. 2007 Mar;177(3):370-83. doi: 10.1007/s00221-006-0681-6.

Effects of mu-opioid receptor modulation on GABAB receptor synaptic function in hippocampal CA1.μ-阿片受体调节对海马CA1区GABAB受体突触功能的影响

J Neurophysiol. 2007 Mar;97(3):2301-11. doi: 10.1152/jn.01179.2006. Epub 2007 Jan 10.

[Hyperexcitability CA1 neurons induced by brief episodes of hypoxia in hippocampal slices in rats of various age groups].[不同年龄组大鼠海马切片短暂缺氧诱导的CA1神经元兴奋性过高]

Ross Fiziol Zh Im I M Sechenova. 2004 Jan;90(1):121-6.

本文引用的文献

Ross Fiziol Zh Im I M Sechenova. 2004 Jan;90(1):121-6.

GABA uptake regulates cortical excitability via cell type-specific tonic inhibition.γ-氨基丁酸（GABA）摄取通过细胞类型特异性的紧张性抑制来调节皮质兴奋性。

Nat Neurosci. 2003 May;6(5):484-90. doi: 10.1038/nn1043.

Network and pharmacological mechanisms leading to epileptiform synchronization in the limbic system in vitro.体外边缘系统中导致癫痫样同步化的网络和药理学机制。

Prog Neurobiol. 2002 Oct;68(3):167-207. doi: 10.1016/s0301-0082(02)00077-1.

Neuronal hyperexcitability induced by repeated brief episodes of hypoxia in rat hippocampal slices: involvement of ionotropic glutamate receptors and L-type Ca(2+) channels.大鼠海马切片中反复短暂缺氧诱导的神经元过度兴奋：离子型谷氨酸受体和L型钙通道的作用

Neuropharmacology. 2002 Mar;42(4):459-66. doi: 10.1016/s0028-3908(02)00005-9.

[Long-term potentiation of glutamatergic transmission and epileptiform activity induced by short-term episodes of anoxia in the CA1 field of the rat hippocampal slices].[大鼠海马切片CA1区短期缺氧发作诱导的谷氨酸能传递长期增强和癫痫样活动]

Ross Fiziol Zh Im I M Sechenova. 2000 Dec;86(12):1676-81.

Effects of postsynaptic GABA(B) receptor activation on epileptiform activity in hippocampal slices.突触后GABA(B)受体激活对海马切片癫痫样活动的影响。

Neuropharmacology. 2001;40(1):131-8. doi: 10.1016/s0028-3908(00)00120-9.

Long-lasting changes in synaptic excitability induced by anoxia in the rat hippocampus.缺氧诱导的大鼠海马体突触兴奋性的长期变化。

Prog Neuropsychopharmacol Biol Psychiatry. 1997 Jan;21(1):211-32. doi: 10.1016/s0278-5846(96)00139-x.

Incidence of epilepsy and unprovoked seizures in Rochester, Minnesota: 1935-1984.明尼苏达州罗切斯特市癫痫和特发性癫痫发作的发病率：1935 - 1984年

Epilepsia. 1993 May-Jun;34(3):453-68. doi: 10.1111/j.1528-1157.1993.tb02586.x.

Synaptic modifications accompanying epileptogenesis in vitro: long-term depression of GABA-mediated inhibition.体外癫痫发生过程中的突触修饰：GABA 介导的抑制性突触后电位的长时程抑制

Brain Res. 1993 Nov 12;627(2):330-40. doi: 10.1016/0006-8993(93)90338-n.

Neonatal hypoxia and epileptic risk: a clinical prospective study.新生儿缺氧与癫痫风险：一项临床前瞻性研究。

Epilepsia. 1984 Apr;25(2):131-6. doi: 10.1111/j.1528-1157.1984.tb04168.x.

文献检索

告别复杂PubMed语法，用中文像聊天一样搜索，搜遍4000万医学文献。AI智能推荐，让科研检索更轻松。

立即免费搜索

文件翻译

保留排版，准确专业，支持PDF/Word/PPT等文件格式，支持 12+语言互译。

免费翻译文档

深度研究

AI帮你快速写综述，25分钟生成高质量综述，智能提取关键信息，辅助科研写作。

立即免费体验