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Pharmacological modulation of PAF: a therapeutic approach to endotoxin shock.

作者信息

Sánchez Crespo M, Fernández-Gallardo S

机构信息

Fundación Jiménez Díaz, Centro Asociado al CSIC, Madrid, Spain.

出版信息

J Lipid Mediat. 1991 Sep-Oct;4(2):127-43.

PMID:1659463
Abstract
摘要

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1
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[Platelet-activating factor (PAF) receptor-related pathophysiology].[血小板活化因子（PAF）受体相关病理生理学]
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引用本文的文献

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Role for intracellular platelet-activating factor in the circulatory failure in a model of gram-positive shock.细胞内血小板活化因子在革兰氏阳性菌感染性休克模型循环衰竭中的作用。
Br J Pharmacol. 1995 Dec;116(8):3191-8. doi: 10.1111/j.1476-5381.1995.tb15123.x.
2
Effect of the platelet-activating factor antagonist, TCV-309, and the cyclo-oxygenase inhibitor, ibuprofen, on the haemodynamic changes in canine experimental endotoxic shock.血小板激活因子拮抗剂TCV - 309和环氧化酶抑制剂布洛芬对犬实验性内毒素休克血流动力学变化的影响。
Br J Pharmacol. 1993 Dec;110(4):1501-7. doi: 10.1111/j.1476-5381.1993.tb13992.x.
3
Are events after endotoxemia related to circulating phospholipase A2?
内毒素血症后的事件与循环中的磷脂酶A2有关吗？
Ann Surg. 1994 Feb;219(2):183-92. doi: 10.1097/00000658-199402000-00010.
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Partial characterization of lipids that develop during the routine storage of blood and prime the neutrophil NADPH oxidase.血液常规储存过程中产生并启动中性粒细胞NADPH氧化酶的脂质的部分特性
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Sepsis and septic shock. II. Treatment.脓毒症与脓毒性休克。二、治疗
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Studies of the effect of a platelet-activating factor antagonist, CL 184,005, in animal models of gram-negative bacterial sepsis.血小板激活因子拮抗剂CL 184,005在革兰氏阴性菌败血症动物模型中的作用研究。
Antimicrob Agents Chemother. 1992 Sep;36(9):1971-7. doi: 10.1128/AAC.36.9.1971.
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More on pathogenesis and treatment of septic shock.关于感染性休克的发病机制与治疗的更多内容。
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