Cleaver J E, Weil S
J Virol. 1975 Jul;16(1):214-6. doi: 10.1128/JVI.16.1.214-216.1975.
Irradiation of a simian virus 40 temperature-sensitive late mutant before infection of CV1 cells produces viruses that are phenotypically wild-type revertants. This probably occurs through suppression rather than true reversion because the revertants are produced at relatively high frequency and still have some properties of the original temperature-sensitive virus.
在感染CV1细胞之前,对猿猴病毒40温度敏感型晚期突变体进行辐照会产生表型为野生型回复体的病毒。这可能是通过抑制而非真正的回复发生的,因为回复体以相对较高的频率产生,并且仍然具有原始温度敏感型病毒的一些特性。