甲硫氨酸刺激苹果组织切片中乙烯的产生。
Stimulation of ethylene production in apple tissue slices by methionine.
机构信息
Pioneering Research Laboratory for Post Harvest Plant Physiology, Plant Industry Station, Beltsville, Maryland.
出版信息
Plant Physiol. 1966 Mar;41(3):376-82. doi: 10.1104/pp.41.3.376.
Methionine can induce more than a 100% increase in ethylene production by apple tissue slices. The increased amount of ethylene derives from carbons 3 and 4 of methionine. Only post-climacteric fruit tissues are stimulated by methionine, and stimulation is optimum after 8 months' storage. Copper chelators such as sodium diethyl dithiocarbamate and cuprizone very markedly inhibit ethylene production by tissue slices. Carbon monoxide does not effect ethylene production by the slices. These data suggest that the mechanism for the conversion of methionine to ethylene, in apple tissues, is similar to the previously described model system for producing ethylene from methionine and reduced copper. Therefore, it is suggested that one of the ethylene-forming systems in tissues derives from methionine and proceeds to ethylene via a copper enzyme system which may be a peroxidase.
蛋氨酸可使苹果组织薄片的乙烯生成量增加 100%以上。增加的乙烯量来自蛋氨酸的第 3 和第 4 位碳原子。只有成熟后的果实组织受蛋氨酸刺激，而且贮藏 8 个月后刺激作用最佳。二乙基二硫代氨基甲酸钠和环烷酸铜等铜螯合剂可非常显著地抑制组织薄片的乙烯生成。一氧化碳对薄片的乙烯生成没有影响。这些资料表明，苹果组织中蛋氨酸转化为乙烯的机制与先前描述的从蛋氨酸和还原铜产生乙烯的模型系统相似。因此，有人认为组织中的一个乙烯形成系统来源于蛋氨酸，并通过一个铜酶系统（可能是过氧化物酶）进行到乙烯。
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