Mitochondrial reactive oxygen species. Contribution to oxidative stress and interorganellar signaling.
作者信息
Rhoads David M, Umbach Ann L, Subbaiah Chalivendra C, Siedow James N
机构信息
School of Life Sciences, Arizona State University, Tempe, 85287-4501, USA.
出版信息
Plant Physiol. 2006 Jun;141(2):357-66. doi: 10.1104/pp.106.079129.
Abstract
摘要
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Arabidopsis nitric oxide synthase1 is targeted to mitochondria and protects against oxidative damage and dark-induced senescence.拟南芥一氧化氮合酶1定位于线粒体，可抵御氧化损伤和黑暗诱导的衰老。
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Ascorbic acid deficiency activates cell death and disease resistance responses in Arabidopsis.抗坏血酸缺乏会激活拟南芥中的细胞死亡和抗病反应。
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In higher plants, only root mitochondria, but not leaf mitochondria reduce nitrite to NO, in vitro and in situ.在高等植物中，仅根部线粒体而非叶片线粒体能够在体外和原位将亚硝酸盐还原为一氧化氮。
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Mitochondria-driven changes in leaf NAD status exert a crucial influence on the control of nitrate assimilation and the integration of carbon and nitrogen metabolism.线粒体驱动的叶片烟酰胺腺嘌呤二核苷酸（NAD）状态变化对硝酸盐同化的控制以及碳氮代谢的整合具有至关重要的影响。
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Sensing and signalling in response to oxygen deprivation in plants and other organisms.植物及其他生物体对缺氧的感知与信号传导
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