父母创伤后应激症状作为儿科损伤患者儿童急性生物学反应及随后创伤后应激症状的调节因素。
Parental posttraumatic stress symptoms as a moderator of child's acute biological response and subsequent posttraumatic stress symptoms in pediatric injury patients.
作者信息
Nugent Nicole R, Ostrowski Sarah, Christopher Norman C, Delahanty Douglas L
机构信息
Department of Psychology, Kent State University, Kent, Ohio 44242, USA.
出版信息
J Pediatr Psychol. 2007 Apr;32(3):309-18. doi: 10.1093/jpepsy/jsl005. Epub 2006 Jun 8.
OBJECTIVE
To examine how parental responses following pediatric injury may influence their child's posttraumatic stress symptoms (PTSS).
METHODS
Heart rate (HR) from 82 pediatric injury patients was measured during emergency medical services (EMSs) transport and following hospital admission. Twelve-hour urinary cortisol levels were assessed upon admission. Child PTSS and parental PTSS and general distress were assessed 6 weeks and 6 months after trauma.
RESULTS
Six-week parental PTSS predicted 6-month child PTSS even after controlling for demographics and general parent distress (DeltaR(2) = .08, p = .03). Parental PTSS moderated the relationship between (a) child cortisol levels and 6-month child PTSS (DeltaR2 = .08, p = .03) and between (b) hospital HR and 6-month child PTSS (DeltaR2 = .09, p = .03).
CONCLUSION
The present findings suggest that parental response to trauma may interact with child acute physiological responses to predict persistent child PTSS.
目的
探讨儿科损伤后父母的反应如何影响其子女的创伤后应激症状（PTSS）。
方法
在紧急医疗服务（EMS）转运期间及入院后，对82名儿科损伤患者的心率（HR）进行测量。入院时评估12小时尿皮质醇水平。在创伤后6周和6个月评估儿童PTSS、父母PTSS和一般困扰情况。
结果
即使在控制了人口统计学因素和父母一般困扰情况后，6周时父母的PTSS仍可预测6个月时儿童的PTSS（ΔR² = 0.08，p = 0.03）。父母的PTSS调节了（a）儿童皮质醇水平与6个月时儿童PTSS之间的关系（ΔR² = 0.08，p = 0.03）以及（b）入院时心率与6个月时儿童PTSS之间的关系（ΔR² = 0.09，p = 0.03）。
结论
目前的研究结果表明，父母对创伤的反应可能与儿童的急性生理反应相互作用，以预测儿童持续的PTSS。
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