Klenerman P, Semmo N
Peter Medawar Building for Pathogen Research, Nuffield Department of Medicine, University of Oxford, Oxford OX3 9TU, UK.
Gut. 2006 Jul;55(7):914-6. doi: 10.1136/gut.2005.087304.
Failure to sustain antiviral T cell responses is a hallmark of persistent hepatitis C virus infection. Early loss of T cell proliferative capacity after acute disease appears to be an important component of this process. T cell function may, however, be restored in vitro with interleukin 2
无法维持抗病毒T细胞应答是丙型肝炎病毒持续感染的一个标志。急性疾病后T细胞增殖能力的早期丧失似乎是这一过程的一个重要组成部分。然而,T细胞功能在体外可通过白细胞介素2恢复。