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Stroke therapy.

作者信息

Zivin J A, Choi D W

机构信息

University of California, San Diego, School of Medicine.

出版信息

Sci Am. 1991 Jul;265(1):56-63. doi: 10.1038/scientificamerican0791-56.

DOI:10.1038/scientificamerican0791-56
PMID:1679258
Abstract
摘要

相似文献

1
Stroke therapy.中风治疗。
Sci Am. 1991 Jul;265(1):56-63. doi: 10.1038/scientificamerican0791-56.
2
Excitatory amino acid receptor antagonists: a novel treatment for ischemic cerebrovascular diseases.兴奋性氨基酸受体拮抗剂：缺血性脑血管疾病的一种新型治疗方法。
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3
Tissue plasminogen activator plus glutamate antagonist improves outcome after embolic stroke.
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4
[Diagnosis, therapy and prevention of cerebrovascular diseases. 1. Clinical aspects and diagnosis].[脑血管疾病的诊断、治疗与预防。1. 临床方面与诊断]
Schweiz Med Wochenschr. 1989 May 13;119(19):613-29.
5
Tissue plasminogen activator reduces neurological damage after cerebral embolism.
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6
Reduction of infarct volume and mortality by thrombolysis in a rat embolic stroke model.在大鼠栓塞性中风模型中，溶栓可减少梗死体积并降低死亡率。
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7
Protection by diphenyliodonium against glutamate neurotoxicity due to blocking of N-methyl-D-aspartate receptors.
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8
Receptor types mediating the rise in the cytosolic free calcium concentration by L-aspartate and L-glutamate in immature cerebellar neurons with N-methyl-D-aspartate receptors.
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9
Progress towards stroke prevention: Robert Wartenberg lecture.
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10
Prevention of post-traumatic excitotoxic brain damage with NMDA antagonist drugs: a new strategy for the nineties.
Acta Neurochir Suppl (Wien). 1992;55:49-55. doi: 10.1007/978-3-7091-9233-7_15.

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Automated multimodal segmentation of acute ischemic stroke lesions on clinical MR images.基于临床磁共振图像的急性缺血性脑卒中病灶的自动多模态分割。
Magn Reson Imaging. 2022 Oct;92:45-57. doi: 10.1016/j.mri.2022.06.001. Epub 2022 Jun 7.
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Long-Term Dynamic Changes of NMDA Receptors Following an Excitotoxic Challenge.兴奋性神经毒性损伤后 NMDA 受体的长期动态变化。
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Editorial: Excitotoxicity Turns 50. The Death That Never Dies.社论：兴奋性毒性五十周年。永不消逝的死亡。
Front Neurosci. 2022 Jan 17;15:831809. doi: 10.3389/fnins.2021.831809. eCollection 2021.
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A Neurotoxic : Glutamate, Calcium, and Zinc in the Excitotoxic Cascade.神经毒性：兴奋性毒性级联反应中的谷氨酸、钙和锌
Front Mol Neurosci. 2020 Nov 26;13:600089. doi: 10.3389/fnmol.2020.600089. eCollection 2020.
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Going the Extra (Synaptic) Mile: Excitotoxicity as the Road Toward Neurodegenerative Diseases.多走（突触）一步：兴奋性毒性作为通往神经退行性疾病之路
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Therapeutic potential of umbilical cord blood stem cells on brain damage of a model of stroke.脐带血干细胞对中风模型脑损伤的治疗潜力
J Cardiovasc Thorac Res. 2011;3(4):117-22. doi: 10.5681/jcvtr.2011.026. Epub 2011 Dec 28.
7
Reduced early hypoxic/ischemic brain damage is associated with increased GLT-1 levels in mice expressing mutant (P301L) human tau.在表达突变型（P301L）人tau蛋白的小鼠中，早期缺氧/缺血性脑损伤的减轻与谷氨酸转运体-1（GLT-1）水平升高有关。
Brain Res. 2009 Jan 9;1247:159-70. doi: 10.1016/j.brainres.2008.10.022. Epub 2008 Nov 1.
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Neuroprotective therapies in stroke.中风的神经保护疗法。
Drugs. 1997;54 Suppl 3:83-8; discussion 88-9. doi: 10.2165/00003495-199700543-00012.
9
Blockade of glutamate excitotoxicity and its clinical applications.
Neurochem Res. 1993 Apr;18(4):479-83. doi: 10.1007/BF00967252.