• 文献检索
  • 文档翻译
  • 深度研究
  • 学术资讯
  • Suppr Zotero 插件Zotero 插件
  • 邀请有礼
  • 套餐&价格
  • 历史记录
应用&插件
Suppr Zotero 插件Zotero 插件浏览器插件Mac 客户端Windows 客户端微信小程序
定价
高级版会员购买积分包购买API积分包
服务
文献检索文档翻译深度研究API 文档MCP 服务
关于我们
关于 Suppr公司介绍联系我们用户协议隐私条款
关注我们

Suppr 超能文献

核心技术专利:CN118964589B侵权必究
粤ICP备2023148730 号-1Suppr @ 2026

文献检索

告别复杂PubMed语法,用中文像聊天一样搜索,搜遍4000万医学文献。AI智能推荐,让科研检索更轻松。

立即免费搜索

文件翻译

保留排版,准确专业,支持PDF/Word/PPT等文件格式,支持 12+语言互译。

免费翻译文档

深度研究

AI帮你快速写综述,25分钟生成高质量综述,智能提取关键信息,辅助科研写作。

立即免费体验

尼日利亚儿童的重度双侧感音神经性听力损失:病因有任何变化吗?

Profound bilateral sensorineural hearing loss in nigerian children: any shift in etiology?

作者信息

Dunmade A D, Segun-Busari S, Olajide T G, Ologe F E

机构信息

University of Ilorin, P.O. Box 6641, Ilorin 240001, Nigeria.

出版信息

J Deaf Stud Deaf Educ. 2007 Winter;12(1):112-8. doi: 10.1093/deafed/enl019. Epub 2006 Sep 6.

DOI:10.1093/deafed/enl019
PMID:16956969
Abstract

Deafness, profound hearing loss, is a global problem. However, the causes of, attitudes toward, and management options for deafness differ considerably from region to region. This study seeks to identify the present causes of profound sensorineural hearing loss in Nigeria, which in our environment is almost synonymous to a life sentence of silence and isolation. This is a retrospective survey of children 15 years and below (M = 6.7 years, SD = 3.2). Of the 115 children included in this study, 64 (55.7%) were males, giving a male:female ratio of 5:4. Age group 1-3 years had the highest proportion of hearing loss, 33 (28.7%), and there was a progressive decline in frequency with advancing age. In about a third (34.8%) of patients, causes were unknown, probably congenital. The main acquired causes were febrile illness (18.3%), measles (13.9%), meningitis (8.7%), mumps (6.9%), or severe birth asphyxia (4.3%). Compared to the findings of two decades ago, we conclude that there is no significant shift yet in the etiology of profound sensorineural hearing loss in our environment.

摘要

耳聋,即严重听力损失,是一个全球性问题。然而,耳聋的病因、人们对其的态度以及管理方案在不同地区存在很大差异。本研究旨在确定尼日利亚严重感音神经性听力损失的当前病因,在我们所处的环境中,这几乎等同于终身沉默和孤立。这是一项针对15岁及以下儿童(平均年龄M = 6.7岁,标准差SD = 3.2)的回顾性调查。本研究纳入的115名儿童中,64名(55.7%)为男性,男女比例为5:4。1 - 3岁年龄组听力损失比例最高,为33名(28.7%),且随着年龄增长频率逐渐下降。约三分之一(34.8%)的患者病因不明,可能为先天性。主要的后天性病因包括发热性疾病(18.3%)、麻疹(13.9%)、脑膜炎(8.7%)、腮腺炎(6.9%)或严重出生窒息(4.3%)。与二十年前的研究结果相比,我们得出结论,在我们所处的环境中,严重感音神经性听力损失的病因尚未发生显著变化。

相似文献

1
Profound bilateral sensorineural hearing loss in nigerian children: any shift in etiology?尼日利亚儿童的重度双侧感音神经性听力损失:病因有任何变化吗?
J Deaf Stud Deaf Educ. 2007 Winter;12(1):112-8. doi: 10.1093/deafed/enl019. Epub 2006 Sep 6.
2
Prevalence and etiology of congenital or early acquired hearing impairment in Eastern Finland.芬兰东部先天性或早期获得性听力障碍的患病率及病因
Int J Pediatr Otorhinolaryngol. 2009 Oct;73(10):1353-7. doi: 10.1016/j.ijporl.2009.06.009. Epub 2009 Jul 18.
3
Sensorineural hearing loss in adults with sickle cell anaemia.镰状细胞贫血成年患者的感音神经性听力损失
Afr J Med Med Sci. 2002 Mar;31(1):21-4.
4
[Sensorineural hearing loss in children starting school. Results of a representative hearing screening study].[刚入学儿童的感音神经性听力损失。一项代表性听力筛查研究的结果]
HNO. 1995 Jan;43(1):31-4.
5
Bilateral sensorineural hearing loss due to mumps.腮腺炎所致双侧感音神经性听力损失
Br J Clin Pract. 1990 Dec;44(12):757-9.
6
Etiological diagnosis of bilateral, sensorineural hearing impairment in a pediatric Greek population.希腊儿科人群双侧感音神经性听力损失的病因诊断
Int J Pediatr Otorhinolaryngol. 2005 Apr;69(4):449-55. doi: 10.1016/j.ijporl.2004.11.007. Epub 2005 Jan 4.
7
Causes of severe to profound deafness in Omani paediatric population.
Int J Pediatr Otorhinolaryngol. 2004 Oct;68(10):1307-13. doi: 10.1016/j.ijporl.2004.05.002.
8
Pattern of childhood deafness in an audiologic centre in Port Harcourt, Nigeria.尼日利亚哈科特港一家听力中心的儿童耳聋模式。
Niger J Med. 2005 Jul-Sep;14(3):307-10.
9
Children with sensorineural hearing loss after passing the newborn hearing screen.新生儿听力筛查通过后的感音神经性听力损失患儿。
JAMA Otolaryngol Head Neck Surg. 2013 Feb;139(2):119-23. doi: 10.1001/jamaoto.2013.1229.
10
[Unilateral hearing loss in childhood. An empirical analysis comparing bilateral hearing loss].[儿童单侧听力损失。一项比较双侧听力损失的实证分析]
Laryngorhinootologie. 2001 Jan;80(1):18-22. doi: 10.1055/s-2001-11025.

引用本文的文献

1
The Assessment of Auditory Function in High-Risk Neonates and Infants Using Brainstem-Evoked Response Audiometry (BERA).使用脑干诱发电位听力测定法(BERA)对高危新生儿和婴儿的听觉功能进行评估。
Cureus. 2024 Aug 12;16(8):e66666. doi: 10.7759/cureus.66666. eCollection 2024 Aug.
2
The Importance of Lassa Fever and Its Disease Management in West Africa.拉沙热的重要性及其在西非的疾病管理。
Viruses. 2024 Feb 7;16(2):266. doi: 10.3390/v16020266.
3
"Association of High Risk Factors and Hearing Impairment in Infants-A Hospital Based Study".
《婴儿高危因素与听力障碍的关联——一项基于医院的研究》
Indian J Otolaryngol Head Neck Surg. 2022 Dec;74(Suppl 3):3933-3938. doi: 10.1007/s12070-021-02760-0. Epub 2021 Jul 14.
4
Combating Lassa Fever in West African Sub-Region: Progress, Challenges, and Future Perspectives.西非分区域抗击拉沙热:进展、挑战和未来展望。
Viruses. 2023 Jan 3;15(1):146. doi: 10.3390/v15010146.
5
Depletion of CD4 and CD8 T Cells Reduces Acute Disease and Is Not Associated with Hearing Loss in ML29-Infected STAT1-/- Mice.CD4和CD8 T细胞的耗竭可减轻急性疾病,且与感染ML29的STAT1-/-小鼠的听力损失无关。
Biomedicines. 2022 Sep 29;10(10):2433. doi: 10.3390/biomedicines10102433.
6
Cochleovestibular findings linked to COVID-19: A scoping review for clinical care planning in South Africa.与 COVID-19 相关的耳蜗前庭发现:南非临床护理计划的范围综述。
S Afr J Commun Disord. 2022 Aug 12;69(2):e1-e12. doi: 10.4102/sajcd.v69i2.899.
7
Aetiologies of profound bilateral sensorineural hearing loss among children in Ekiti State, South Western Nigeria.尼日利亚西南部埃基提州儿童深度双侧感觉神经性听力损失的病因。
Pan Afr Med J. 2021 Jan 28;38:98. doi: 10.11604/pamj.2021.38.98.21438. eCollection 2021.
8
Embryonic Origins of Virus-Induced Hearing Loss: Overview of Molecular Etiology.病毒诱导性听力损失的胚胎起源:分子病因学概述。
Viruses. 2021 Jan 6;13(1):71. doi: 10.3390/v13010071.
9
Pathological and clinical profile of hearing loss among Sudanese children attending the Khartoum Teaching Hospital.喀土穆教学医院就诊的苏丹儿童听力损失的病理和临床特征
J Family Med Prim Care. 2020 Jun 30;9(6):2720-2723. doi: 10.4103/jfmpc.jfmpc_348_20. eCollection 2020 Jun.
10
Animal Models of Lassa Fever.拉沙热的动物模型
Pathogens. 2020 Mar 6;9(3):197. doi: 10.3390/pathogens9030197.