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DNA polymerase beta null mouse embryonic fibroblasts harbor a homozygous null mutation in DNA polymerase iota.DNA聚合酶β基因敲除小鼠胚胎成纤维细胞在DNA聚合酶ι中存在纯合无效突变。
DNA Repair (Amst). 2007 Jan 4;6(1):3-7. doi: 10.1016/j.dnarep.2006.08.005. Epub 2006 Sep 18.
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DNA polymerase lambda mediates a back-up base excision repair activity in extracts of mouse embryonic fibroblasts.DNA聚合酶λ介导小鼠胚胎成纤维细胞提取物中的一种备用碱基切除修复活性。
J Biol Chem. 2005 May 6;280(18):18469-75. doi: 10.1074/jbc.M411864200. Epub 2005 Mar 3.
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Negligible impact of pol iota expression on the alkylation sensitivity of pol beta-deficient mouse fibroblast cells.聚合酶ι表达对聚合酶β缺陷型小鼠成纤维细胞烷基化敏感性的影响可忽略不计。
DNA Repair (Amst). 2008 Jun 1;7(6):830-3. doi: 10.1016/j.dnarep.2008.02.013. Epub 2008 Apr 22.
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UV-B radiation induces epithelial tumors in mice lacking DNA polymerase eta and mesenchymal tumors in mice deficient for DNA polymerase iota.紫外线B辐射在缺乏DNA聚合酶η的小鼠中诱发上皮肿瘤，在缺乏DNA聚合酶ι的小鼠中诱发间充质肿瘤。
Mol Cell Biol. 2006 Oct;26(20):7696-706. doi: 10.1128/MCB.01076-06. Epub 2006 Aug 5.
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[Activity of error-prone DNA polymerase iota in different periods of house mouse Mus musculus ontogeny].
Ontogenez. 2008 Sep-Oct;39(5):367-73.
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Cutting edge: DNA polymerases mu and lambda are dispensable for Ig gene hypermutation.前沿：DNA聚合酶μ和λ对于Ig基因超突变并非必需。
J Immunol. 2002 Apr 15;168(8):3702-6. doi: 10.4049/jimmunol.168.8.3702.
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Bypass of a psoralen DNA interstrand cross-link by DNA polymerases β, ι, and κ in vitro.体外 DNA 聚合酶 β、ι 和 κ 对补骨脂素 DNA 链间交联的绕过。
Biochemistry. 2012 Nov 6;51(44):8931-8. doi: 10.1021/bi3008565. Epub 2012 Oct 29.
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Arginine methylation regulates DNA polymerase beta.精氨酸甲基化调节DNA聚合酶β。
Mol Cell. 2006 Apr 7;22(1):51-62. doi: 10.1016/j.molcel.2006.02.013.
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129-Derived Mouse Strains Express an Unstable but Catalytically Active DNA Polymerase Iota Variant.源自129的小鼠品系表达一种不稳定但具有催化活性的DNA聚合酶ι变体。
Mol Cell Biol. 2015 Sep 1;35(17):3059-70. doi: 10.1128/MCB.00371-15. Epub 2015 Jun 29.
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Normal hypermutation in antibody genes from congenic mice defective for DNA polymerase iota.来自DNA聚合酶ι缺陷的同源小鼠抗体基因的正常超突变。
DNA Repair (Amst). 2006 Mar 7;5(3):392-8. doi: 10.1016/j.dnarep.2005.12.006. Epub 2006 Jan 26.
引用本文的文献
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Exploiting DNA Endonucleases to Advance Mechanisms of DNA Repair.利用DNA核酸内切酶推进DNA修复机制
Biology (Basel). 2021 Jun 14;10(6):530. doi: 10.3390/biology10060530.
2
Relating Essential Proteins to Drug Side-Effects Using Canonical Component Analysis: A Structure-Based Approach.使用典型成分分析将必需蛋白质与药物副作用相关联：一种基于结构的方法。
J Chem Inf Model. 2015 Jul 27;55(7):1483-94. doi: 10.1021/acs.jcim.5b00030. Epub 2015 Jul 16.
3
HSP90 regulates DNA repair via the interaction between XRCC1 and DNA polymerase β.热休克蛋白90（HSP90）通过X射线修复交叉互补蛋白1（XRCC1）与DNA聚合酶β之间的相互作用来调节DNA修复。
Nat Commun. 2014 Nov 26;5:5513. doi: 10.1038/ncomms6513.
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Transcriptional profiling reveals elevated Sox2 in DNA polymerase ß null mouse embryonic fibroblasts.转录谱分析揭示了 DNA 聚合酶 β 缺失型小鼠胚胎成纤维细胞中 Sox2 的升高。
Am J Cancer Res. 2012;2(6):699-713. doi: 10.1158/1538-7445.am2012-699. Epub 2012 Nov 20.
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A germline polymorphism of DNA polymerase beta induces genomic instability and cellular transformation.DNA 聚合酶 β 的种系多态性诱导基因组不稳定性和细胞转化。
PLoS Genet. 2012;8(11):e1003052. doi: 10.1371/journal.pgen.1003052. Epub 2012 Nov 8.
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Deregulated expression of DNA polymerase β is involved in the progression of genomic instability.DNA 聚合酶 β 的失调表达与基因组不稳定性的进展有关。
Environ Mol Mutagen. 2012 Jun;53(5):325-33. doi: 10.1002/em.21697. Epub 2012 May 10.
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Targeting DNA polymerase ß for therapeutic intervention.针对 DNA 聚合酶 ß 的治疗干预。
Curr Mol Pharmacol. 2012 Jan;5(1):68-87.
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Nucleosome disruption by DNA ligase III-XRCC1 promotes efficient base excision repair.DNA 连接酶 III-XRCC1 破坏核小体可促进有效的碱基切除修复。
Mol Cell Biol. 2011 Nov;31(22):4623-32. doi: 10.1128/MCB.05715-11. Epub 2011 Sep 19.
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The base excision repair pathway is required for efficient lentivirus integration.碱基切除修复途径是有效整合慢病毒所必需的。
PLoS One. 2011 Mar 23;6(3):e17862. doi: 10.1371/journal.pone.0017862.
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DNA polymerases beta and lambda mediate overlapping and independent roles in base excision repair in mouse embryonic fibroblasts.DNA 聚合酶β和λ在小鼠胚胎成纤维细胞的碱基切除修复中发挥重叠且独立的作用。
PLoS One. 2010 Aug 18;5(8):e12229. doi: 10.1371/journal.pone.0012229.
本文引用的文献
1
Structure-function studies of DNA polymerase lambda.DNA聚合酶λ的结构-功能研究
DNA Repair (Amst). 2005 Dec 8;4(12):1358-67. doi: 10.1016/j.dnarep.2005.09.001. Epub 2005 Oct 4.
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The role of base excision repair in the sensitivity and resistance to temozolomide-mediated cell death.碱基切除修复在对替莫唑胺介导的细胞死亡的敏感性和抗性中的作用。
Cancer Res. 2005 Jul 15;65(14):6394-400. doi: 10.1158/0008-5472.CAN-05-0715.
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DNA polymerase lambda protects mouse fibroblasts against oxidative DNA damage and is recruited to sites of DNA damage/repair.DNA聚合酶λ保护小鼠成纤维细胞免受氧化性DNA损伤，并被招募至DNA损伤/修复位点。
J Biol Chem. 2005 Sep 9;280(36):31641-7. doi: 10.1074/jbc.C500256200. Epub 2005 Jul 7.
4
REV1 mediated mutagenesis in base excision repair deficient mouse fibroblast.REV1介导的碱基切除修复缺陷型小鼠成纤维细胞中的诱变作用。
DNA Repair (Amst). 2005 Sep 28;4(10):1182-8. doi: 10.1016/j.dnarep.2005.05.002.
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DNA polymerase lambda mediates a back-up base excision repair activity in extracts of mouse embryonic fibroblasts.DNA聚合酶λ介导小鼠胚胎成纤维细胞提取物中的一种备用碱基切除修复活性。
J Biol Chem. 2005 May 6;280(18):18469-75. doi: 10.1074/jbc.M411864200. Epub 2005 Mar 3.
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Poly(ADP-ribose) polymerase activity prevents signaling pathways for cell cycle arrest after DNA methylating agent exposure.聚（ADP - 核糖）聚合酶活性可阻止DNA甲基化剂暴露后细胞周期停滞的信号通路。
J Biol Chem. 2005 Apr 22;280(16):15773-85. doi: 10.1074/jbc.M413841200. Epub 2005 Feb 7.
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Genetic interaction between DNA polymerase beta and DNA-PKcs in embryogenesis and neurogenesis.胚胎发育和神经发生过程中DNA聚合酶β与DNA-PKcs之间的基因相互作用。
Cell Death Differ. 2005 Feb;12(2):184-91. doi: 10.1038/sj.cdd.4401543.
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'Knock down' of DNA polymerase beta by RNA interference: recapitulation of null phenotype.通过RNA干扰“敲低”DNA聚合酶β：无效表型的重现
DNA Repair (Amst). 2004 Nov 2;3(11):1469-74. doi: 10.1016/j.dnarep.2004.05.011.
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Imbalanced base excision repair in response to folate deficiency is accelerated by polymerase beta haploinsufficiency.聚合酶β单倍体不足会加速因叶酸缺乏而导致的碱基切除修复失衡。
J Biol Chem. 2004 Aug 27;279(35):36504-13. doi: 10.1074/jbc.M405185200. Epub 2004 Jun 24.
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