Berjon J J, Munuera L, Calvo M
Department of Traumatology and Orthopedic Surgery, Ramon y Cajal Hospital, Madrid, Spain.
Skeletal Radiol. 1990;19(8):567-74. doi: 10.1007/BF00241278.
Meniscal repair was studied to evaluate the mechanism and its potential protective effects on the articular cartilage in an experimental model consisting of 68 knees of adult dogs on which five different types of medial meniscectomy were performed. The results were assessed by macroscopic, microangiographic, and histological methods, after a sequential follow-up period of 10-450 days. Two different mechanisms of meniscal repair were observed, depending on whether meniscal section had been performed in vascular (total meniscectomy) or avascular (subtotal or partial meniscectomy) zones. It was also observed that the repaired meniscal tissue does not prevent articular cartilage degeneration. This is more closely related to the size of the meniscal fragment preserved at meniscectomy. Due to the biomechanical importance of the meniscus and the lack of functional relevance of the repaired meniscal tissue, the most conservative approach possible to meniscectomy is recommended.
在一个由68只成年犬膝关节组成的实验模型中,对半月板修复进行了研究,该模型进行了五种不同类型的内侧半月板切除术,以评估其机制及其对关节软骨的潜在保护作用。在10 - 450天的连续随访期后,通过宏观、微血管造影和组织学方法评估结果。观察到两种不同的半月板修复机制,这取决于半月板切除术是在血管区(全半月板切除术)还是无血管区(次全或部分半月板切除术)进行。还观察到修复的半月板组织并不能防止关节软骨退变。这与半月板切除术中保留的半月板碎片大小关系更为密切。由于半月板的生物力学重要性以及修复的半月板组织缺乏功能相关性,建议对半月板切除术采用尽可能保守的方法。