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永久性心室和心房起搏患者的左心房大小及壁运动

Left atrial size and wall motion in patients with permanent ventricular and atrial pacing.

作者信息

Kubica J, Stolarczyk L, Krzyminska E, Krasowski R, Raczak G, Lubiński A, Stanke A, Swiatecka G

机构信息

III Department of Internal Medicine, Medical Academy of Gdańsk, Poland.

出版信息

Pacing Clin Electrophysiol. 1990 Dec;13(12 Pt 2):1737-41. doi: 10.1111/j.1540-8159.1990.tb06882.x.

Abstract

It is well known that during permanent ventricular pacing atrial arrhythmias and embolic complications occur much more frequently in comparison to permanent atrial or sequential pacing. Hemodynamic disturbances caused by ventriculoatrial conduction (VAC) are thought to be responsible for those complications. The aim of this study was to compare the left atrial size and its wall motion in three groups of patients with sick sinus syndrome. Group 1: 58 patients with VVI pacing and VAC observed (22 males, 36 females, aged 31-86, mean 62.3). Group 2: 43 patients with primary AAI pacing (13 males, 30 females, aged 27-74, mean 57.8). Group 3: 13 patients with AAI or DDD replacing the primary VVI mode due to pacemaker syndrome and/or heart failure, all with VAC present during VVI pacing (7 males, 6 females, aged 26-80, mean 59.8). Two-dimensional/M-mode echocardiography was performed in all these patients. In group 1 mean diastolic as well as mean systolic atrial diameters were significantly greater (P less than 0.005) and wall motion significantly smaller (P less than 0.005) in comparison to the other groups. Left atrial wall motion amounted to only 7.4% of the mean diastolic diameter in this group. Mean left atrial diastolic and systolic diameters and wall motion in patients with pacemakers preserving atrioventricular synchrony (group 2 and group 3) were almost identical and wall motion amounted to about 22% of the diastolic diameter in both these groups. We conclude that ventriculoatrial conduction leads to significant enlargement of left atrium and to the atrial wall-motion decrease. This predisposes to arrhythmias and embolic complications.(ABSTRACT TRUNCATED AT 250 WORDS)

摘要

众所周知,与永久性心房起搏或顺序起搏相比,永久性心室起搏时房性心律失常和栓塞并发症的发生频率要高得多。室房传导(VAC)引起的血流动力学紊乱被认为是这些并发症的原因。本研究的目的是比较三组病态窦房结综合征患者的左心房大小及其壁运动。第一组:58例观察到VVI起搏和VAC的患者(男22例,女36例,年龄31 - 86岁,平均62.3岁)。第二组:43例原发性AAI起搏患者(男13例,女30例,年龄27 - 74岁,平均57.8岁)。第三组:13例因起搏器综合征和/或心力衰竭而将原发性VVI模式替换为AAI或DDD的患者,所有患者在VVI起搏时均存在VAC(男7例,女6例,年龄26 - 80岁,平均59.8岁)。所有这些患者均进行了二维/M型超声心动图检查。与其他组相比,第一组的平均舒张期以及平均收缩期心房直径显著更大(P小于0.005),壁运动显著更小(P小于0.005)。该组左心房壁运动仅占平均舒张期直径的7.4%。保持房室同步的起搏器患者(第二组和第三组)的平均左心房舒张期和收缩期直径以及壁运动几乎相同,且这两组的壁运动均约占舒张期直径的22%。我们得出结论,室房传导导致左心房显著扩大和心房壁运动减少。这易引发心律失常和栓塞并发症。(摘要截短于250字)

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