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本文引用的文献

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Staphyloxanthin plays a role in the fitness of Staphylococcus aureus and its ability to cope with oxidative stress.金黄色葡萄球菌黄素在金黄色葡萄球菌的适应性及其应对氧化应激的能力中发挥作用。
Infect Immun. 2006 Aug;74(8):4950-3. doi: 10.1128/IAI.00204-06.
2
The presence of peptidoglycan O-acetyltransferase in various staphylococcal species correlates with lysozyme resistance and pathogenicity.各种葡萄球菌属物种中肽聚糖O - 乙酰转移酶的存在与溶菌酶抗性和致病性相关。
Infect Immun. 2006 Aug;74(8):4598-604. doi: 10.1128/IAI.00301-06.
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Activity of the major staphylococcal autolysin Atl.主要葡萄球菌自溶素Atl的活性
FEMS Microbiol Lett. 2006 Jun;259(2):260-8. doi: 10.1111/j.1574-6968.2006.00281.x.
4
In vivo protective role of human group IIa phospholipase A2 against experimental anthrax.人IIa型磷脂酶A2对实验性炭疽的体内保护作用
J Immunol. 2005 Nov 15;175(10):6786-91. doi: 10.4049/jimmunol.175.10.6786.
5
The interactions of antimicrobial peptides derived from lysozyme with model membrane systems.源自溶菌酶的抗菌肽与模型膜系统的相互作用。
Biochim Biophys Acta. 2005 Mar 1;1668(2):175-89. doi: 10.1016/j.bbamem.2004.12.004. Epub 2005 Jan 26.
6
Why are pathogenic staphylococci so lysozyme resistant? The peptidoglycan O-acetyltransferase OatA is the major determinant for lysozyme resistance of Staphylococcus aureus.为什么致病性葡萄球菌对溶菌酶如此耐药？肽聚糖O-乙酰基转移酶OatA是金黄色葡萄球菌对溶菌酶耐药的主要决定因素。
Mol Microbiol. 2005 Feb;55(3):778-87. doi: 10.1111/j.1365-2958.2004.04446.x.
7
Role of teichoic acids in Staphylococcus aureus nasal colonization, a major risk factor in nosocomial infections.磷壁酸在金黄色葡萄球菌鼻腔定植中的作用，医院感染的主要危险因素。
Nat Med. 2004 Mar;10(3):243-5. doi: 10.1038/nm991. Epub 2004 Feb 1.
8
ON THE LINKAGE BETWEEN TEICHOIC ACID AND THE GLYCOPEPTIDE IN THE CELL WALL OF STAPHYLOCOCCUS AUREUS.关于金黄色葡萄球菌细胞壁中磷壁酸与糖肽之间的联系
Biochem Biophys Res Commun. 1963 Aug 14;12:418-24. doi: 10.1016/0006-291x(63)90117-7.
9
Increased expression of antimicrobial peptides and lysozyme in colonic epithelial cells of patients with ulcerative colitis.溃疡性结肠炎患者结肠上皮细胞中抗菌肽和溶菌酶表达增加。
Clin Exp Immunol. 2003 Jan;131(1):90-101. doi: 10.1046/j.1365-2249.2003.02035.x.
10
Staphylococcus aureus strains lacking D-alanine modifications of teichoic acids are highly susceptible to human neutrophil killing and are virulence attenuated in mice.缺乏磷壁酸D-丙氨酸修饰的金黄色葡萄球菌菌株对人类中性粒细胞杀伤高度敏感，并且在小鼠中毒力减弱。
J Infect Dis. 2002 Jul 15;186(2):214-9. doi: 10.1086/341454. Epub 2002 Jul 3.

壁磷壁酸对金黄色葡萄球菌溶菌酶抗性的影响。

Influence of wall teichoic acid on lysozyme resistance in Staphylococcus aureus.

作者信息

Bera Agnieszka, Biswas Raja, Herbert Silvia, Kulauzovic Emir, Weidenmaier Christopher, Peschel Andreas, Götz Friedrich

机构信息

Microbial Genetics, University of Tübingen, 72076 Tübingen, Germany.

出版信息

J Bacteriol. 2007 Jan;189(1):280-3. doi: 10.1128/JB.01221-06. Epub 2006 Nov 3.

DOI:10.1128/JB.01221-06
PMID:17085565
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC1797201/
Abstract

Staphylococcus aureus peptidoglycan (PG) is completely resistant to the hydrolytic activity of lysozyme. Here we show that modifications in PG by O acetylation, wall teichoic acid, and a high degree of cross-linking contribute to this resistance.

摘要

金黄色葡萄球菌肽聚糖（PG）对溶菌酶的水解活性完全具有抗性。在此我们表明，通过O-乙酰化、壁磷壁酸和高度交联对PG进行的修饰促成了这种抗性。