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1
"Oncogenic shock": explaining oncogene addiction through differential signal attenuation.
Clin Cancer Res. 2006 Jul 15;12(14 Pt 2):4392s-4395s. doi: 10.1158/1078-0432.CCR-06-0096.
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EGF receptor gene mutations are common in lung cancers from "never smokers" and are associated with sensitivity of tumors to gefitinib and erlotinib.
Proc Natl Acad Sci U S A. 2004 Sep 7;101(36):13306-11. doi: 10.1073/pnas.0405220101. Epub 2004 Aug 25.
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EGFR mutations in lung cancer: correlation with clinical response to gefitinib therapy.
Science. 2004 Jun 4;304(5676):1497-500. doi: 10.1126/science.1099314. Epub 2004 Apr 29.
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Activating mutations in the epidermal growth factor receptor underlying responsiveness of non-small-cell lung cancer to gefitinib.
N Engl J Med. 2004 May 20;350(21):2129-39. doi: 10.1056/NEJMoa040938. Epub 2004 Apr 29.
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JAK/STAT, Raf/MEK/ERK, PI3K/Akt and BCR-ABL in cell cycle progression and leukemogenesis.
Leukemia. 2004 Feb;18(2):189-218. doi: 10.1038/sj.leu.2403241.
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Janus faces of ras: anti or pro-apoptotic?
Apoptosis. 1999 Oct;4(5):383-8. doi: 10.1023/a:1009651406017.

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