肝细胞再生和肝小叶修复在肝损伤最终结局中的作用。一种两阶段毒性模型。 - Suppr

Suppr

超能文献

Role of hepatocellular regeneration and hepatolobular healing in the final outcome of liver injury. A two-stage model of toxicity.

作者信息

Mehendale H M

机构信息

Department of Pharmacology and Toxicology, University of Mississippi Medical Center, Jackson 39216-4505.

出版信息

Biochem Pharmacol. 1991 Aug 22;42(6):1155-62. doi: 10.1016/0006-2952(91)90249-5.

DOI:10.1016/0006-2952(91)90249-5

PMID:1716097

Abstract

摘要

相似文献

Role of hepatocellular regeneration and hepatolobular healing in the final outcome of liver injury. A two-stage model of toxicity.

Biochem Pharmacol. 1991 Aug 22;42(6):1155-62. doi: 10.1016/0006-2952(91)90249-5.

Bromotrichloromethane hepatotoxicity. The role of stimulated hepatocellular regeneration in recovery: biochemical and histopathological studies in control and chlordecone pretreated male rats.

Toxicol Pathol. 1990;18(4 Pt 2):667-77. doi: 10.1177/019262339001800426.

Hepatotoxicity and lethality of halomethanes in Mongolian gerbils pretreated with chlordecone, phenobarbital or mirex.

Arch Toxicol. 1991;65(3):204-12. doi: 10.1007/BF02307310.

Potentiation of halomethane hepatotoxicity by chlordecone: a hypothesis for the mechanism.

Med Hypotheses. 1990 Dec;33(4):289-99. doi: 10.1016/0306-9877(90)90144-4.

Phospholipase A2 activation and cell injury in isolated rat hepatocytes exposed to bromotrichloromethane, chloroform, and 1,1-dichloroethylene as compared to effects of carbon tetrachloride.

Toxicol Appl Pharmacol. 1992 Mar;113(1):159-62. doi: 10.1016/0041-008x(92)90021-j.

Colchicine antimitosis abolishes resiliency of postnatally developing rats to chlordecone-amplified carbon tetrachloride hepatotoxicity and lethality.

Environ Health Perspect. 1998 Sep;106(9):597-606. doi: 10.1289/ehp.98106597.

Pivotal role of hepatocellular regeneration in the ultimate hepatotoxicity of CCl4 in chlordecone-, mirex-, or phenobarbital-pretreated rats.

Toxicol Pathol. 1992;20(4):556-69. doi: 10.1177/019262339202000402.

Biochemical assessment of the genotoxicity of the in vitro interaction between chlordecone and carbon tetrachloride in rat hepatocytes.

J Appl Toxicol. 1991 Aug;11(4):303-10. doi: 10.1002/jat.2550110413.

Chlorinated methanes and liver injury: highlights of the past 50 years.

Annu Rev Pharmacol Toxicol. 2000;40:42-65. doi: 10.1146/annurev.pharmtox.40.1.43.

In vivo proton nuclear magnetic resonance imaging and spectroscopy studies of halocarbon-induced liver damage.

Magn Reson Med. 1989 Feb;9(2):229-39. doi: 10.1002/mrm.1910090208.

引用本文的文献

The application of cytokeratin-18 as a biomarker for drug-induced liver injury.

Arch Toxicol. 2021 Nov;95(11):3435-3448. doi: 10.1007/s00204-021-03121-0. Epub 2021 Jul 29.

Protective Effects of Mill. Fruit on Carbon Tetrachloride-induced Hepatotoxicity Mediated through Mitochondria and Restoration of Cellular Energy Content.

Iran J Pharm Res. 2020 Fall;19(4):354-364. doi: 10.22037/ijpr.2020.112534.13812.

Comparison of liver regeneration after partial hepatectomy and acetaminophen-induced acute liver failure: A global picture based on transcriptome analysis.

Food Chem Toxicol. 2020 May;139:111186. doi: 10.1016/j.fct.2020.111186. Epub 2020 Feb 8.

Disruption of Estrogen Receptor Alpha in Rats Results in Faster Initiation of Compensatory Regeneration Despite Higher Liver Injury After Carbon Tetrachloride Treatment.

Int J Toxicol. 2017 May/Jun;36(3):199-206. doi: 10.1177/1091581817706067. Epub 2017 May 8.

Wnt/β-Catenin Signaling Drives Thioacetamide-Mediated Heteroprotection Against Acetaminophen-Induced Lethal Liver Injury.

Dose Response. 2017 Jan 30;15(1):1559325817690287. doi: 10.1177/1559325817690287. eCollection 2017 Jan-Mar.

Dietary restriction (DR) and its advantages.

Indian J Clin Biochem. 2004 Jan;19(1):1-5. doi: 10.1007/BF02872379.

Evaluating pharmacokinetic and pharmacodynamic interactions with computational models in supporting cumulative risk assessment.

Int J Environ Res Public Health. 2011 May;8(5):1613-30. doi: 10.3390/ijerph8051613. Epub 2011 May 19.

Acetaminophen-NAPQI hepatotoxicity: a cell line model system genome-wide association study.

Toxicol Sci. 2011 Mar;120(1):33-41. doi: 10.1093/toxsci/kfq375. Epub 2010 Dec 22.

Role of tissue repair in toxicologic interactions among hepatotoxic organics.

Environ Health Perspect. 1998 Dec;106 Suppl 6(Suppl 6):1307-17. doi: 10.1289/ehp.98106s61307.

Effect of an antimitotic agent colchicine on thioacetamide hepatotoxicity.

Environ Health Perspect. 1996 Jul;104(7):744-9. doi: 10.1289/ehp.96104744.

文献AI研究员

20分钟写一篇综述，助力文献阅读效率提升50倍。

立即体验

用中文搜PubMed

大模型驱动的PubMed中文搜索引擎

马上搜索

文档翻译

学术文献翻译模型，支持多种主流文档格式。

立即体验

Role of hepatocellular regeneration and hepatolobular healing in the final outcome of liver injury. A two-stage model of toxicity.

作者信息

Mehendale H M

机构信息

Department of Pharmacology and Toxicology, University of Mississippi Medical Center, Jackson 39216-4505.

出版信息

Biochem Pharmacol. 1991 Aug 22;42(6):1155-62. doi: 10.1016/0006-2952(91)90249-5.

DOI:10.1016/0006-2952(91)90249-5

PMID:1716097

Abstract

摘要

相似文献

Role of hepatocellular regeneration and hepatolobular healing in the final outcome of liver injury. A two-stage model of toxicity.

Biochem Pharmacol. 1991 Aug 22;42(6):1155-62. doi: 10.1016/0006-2952(91)90249-5.

Bromotrichloromethane hepatotoxicity. The role of stimulated hepatocellular regeneration in recovery: biochemical and histopathological studies in control and chlordecone pretreated male rats.

Toxicol Pathol. 1990;18(4 Pt 2):667-77. doi: 10.1177/019262339001800426.

Hepatotoxicity and lethality of halomethanes in Mongolian gerbils pretreated with chlordecone, phenobarbital or mirex.

Arch Toxicol. 1991;65(3):204-12. doi: 10.1007/BF02307310.

Potentiation of halomethane hepatotoxicity by chlordecone: a hypothesis for the mechanism.

Med Hypotheses. 1990 Dec;33(4):289-99. doi: 10.1016/0306-9877(90)90144-4.

Phospholipase A2 activation and cell injury in isolated rat hepatocytes exposed to bromotrichloromethane, chloroform, and 1,1-dichloroethylene as compared to effects of carbon tetrachloride.

Toxicol Appl Pharmacol. 1992 Mar;113(1):159-62. doi: 10.1016/0041-008x(92)90021-j.

Colchicine antimitosis abolishes resiliency of postnatally developing rats to chlordecone-amplified carbon tetrachloride hepatotoxicity and lethality.

Environ Health Perspect. 1998 Sep;106(9):597-606. doi: 10.1289/ehp.98106597.

Pivotal role of hepatocellular regeneration in the ultimate hepatotoxicity of CCl4 in chlordecone-, mirex-, or phenobarbital-pretreated rats.

Toxicol Pathol. 1992;20(4):556-69. doi: 10.1177/019262339202000402.

Biochemical assessment of the genotoxicity of the in vitro interaction between chlordecone and carbon tetrachloride in rat hepatocytes.

J Appl Toxicol. 1991 Aug;11(4):303-10. doi: 10.1002/jat.2550110413.

Chlorinated methanes and liver injury: highlights of the past 50 years.

Annu Rev Pharmacol Toxicol. 2000;40:42-65. doi: 10.1146/annurev.pharmtox.40.1.43.

In vivo proton nuclear magnetic resonance imaging and spectroscopy studies of halocarbon-induced liver damage.

Magn Reson Med. 1989 Feb;9(2):229-39. doi: 10.1002/mrm.1910090208.

引用本文的文献

The application of cytokeratin-18 as a biomarker for drug-induced liver injury.

Arch Toxicol. 2021 Nov;95(11):3435-3448. doi: 10.1007/s00204-021-03121-0. Epub 2021 Jul 29.

Protective Effects of Mill. Fruit on Carbon Tetrachloride-induced Hepatotoxicity Mediated through Mitochondria and Restoration of Cellular Energy Content.

Iran J Pharm Res. 2020 Fall;19(4):354-364. doi: 10.22037/ijpr.2020.112534.13812.

Comparison of liver regeneration after partial hepatectomy and acetaminophen-induced acute liver failure: A global picture based on transcriptome analysis.

Food Chem Toxicol. 2020 May;139:111186. doi: 10.1016/j.fct.2020.111186. Epub 2020 Feb 8.

Disruption of Estrogen Receptor Alpha in Rats Results in Faster Initiation of Compensatory Regeneration Despite Higher Liver Injury After Carbon Tetrachloride Treatment.

Int J Toxicol. 2017 May/Jun;36(3):199-206. doi: 10.1177/1091581817706067. Epub 2017 May 8.

Wnt/β-Catenin Signaling Drives Thioacetamide-Mediated Heteroprotection Against Acetaminophen-Induced Lethal Liver Injury.

Dose Response. 2017 Jan 30;15(1):1559325817690287. doi: 10.1177/1559325817690287. eCollection 2017 Jan-Mar.

Dietary restriction (DR) and its advantages.

Indian J Clin Biochem. 2004 Jan;19(1):1-5. doi: 10.1007/BF02872379.

Evaluating pharmacokinetic and pharmacodynamic interactions with computational models in supporting cumulative risk assessment.

Int J Environ Res Public Health. 2011 May;8(5):1613-30. doi: 10.3390/ijerph8051613. Epub 2011 May 19.

Acetaminophen-NAPQI hepatotoxicity: a cell line model system genome-wide association study.

Toxicol Sci. 2011 Mar;120(1):33-41. doi: 10.1093/toxsci/kfq375. Epub 2010 Dec 22.

Role of tissue repair in toxicologic interactions among hepatotoxic organics.

Environ Health Perspect. 1998 Dec;106 Suppl 6(Suppl 6):1307-17. doi: 10.1289/ehp.98106s61307.

Effect of an antimitotic agent colchicine on thioacetamide hepatotoxicity.

Environ Health Perspect. 1996 Jul;104(7):744-9. doi: 10.1289/ehp.96104744.