Bakhtiary Farhad, Dzemali Omer, Bastanier Christian K, Moritz Anton, Kleine Peter
Department of Thoracic and Cardiovascular Surgery of Johann Wolfgang Goethe University Hospital, Theodor-Stern-Kai 7, 60596 Frankfurt/Main, Germany.
Europace. 2007 Feb;9(2):94-7. doi: 10.1093/europace/eul172. Epub 2007 Jan 16.
Young children suffering from congenital or post-operative AV-block require life-long pacemaker stimulation. Due to the anatomical prerequisites initially epicardial electrodes are implanted and the generator is placed in the upper abdominal wall. The following study investigated modes of failure leading to reoperation in this group of technically challenging patients.
Between October 2000 and May 2005, a total of 21 infants (age 3 days to 5 years) underwent pacemaker implantation using a subxyphoidal incision for newborns (and a partial lower or complete median sternotomy for older children). Nine patients had previous cardiac surgery for complex congenital defects. The remaining 12 young children suffered from congenital AV-Block (CAVB). Twenty-one bipolar epicardial electrodes (Medtronic Capsure epi) were fixed to the right ventricle, 15 had additional implantation of a bipolar atrial lead. The pacemaker generator (Medtronic Kappa 701) was implanted into the right upper abdominal wall. Indications for revision were recorded. No mortality was observed; pacing and sensing parameters remained stable up to a 5-year follow-up. A total of four reoperations occurred. Three of the four revisions were caused by ventricular electrode fracture. At revision, two electrodes were broken at the crossing between the pericardial cavity and the abdominal wall, one bipolar lead at the Y-division into the two tip electrodes. One reoperation was due to a pacemaker recall. All revisions were performed without complications.
In our institute epicardial pacing in young children was associated with a satisfactory clinical outcome, but also a significant number of failures leading to reoperation, mainly due to electrode fracture caused by the muscular activity of this patient group. Reoperations were performed at a low risk.
患有先天性或术后房室传导阻滞的幼儿需要终身起搏器刺激。由于解剖学上的先决条件,最初植入心外膜电极,起搏器发生器置于上腹壁。以下研究调查了导致这组技术上具有挑战性的患者再次手术的故障模式。
2000年10月至2005年5月期间,共有21名婴儿(年龄3天至5岁)接受了起搏器植入手术,新生儿采用剑突下切口(年龄较大的儿童采用部分低位或完全正中胸骨切开术)。9名患者曾因复杂先天性缺陷接受过心脏手术。其余12名幼儿患有先天性房室传导阻滞(CAVB)。将21根双极心外膜电极(美敦力Capsure epi)固定于右心室,15根额外植入了双极心房导线。起搏器发生器(美敦力Kappa 701)植入右上腹壁。记录再次手术的指征。未观察到死亡病例;直至5年随访期,起搏和感知参数均保持稳定。共进行了4次再次手术。4次翻修中有3次是由心室电极断裂引起的。翻修时,2根电极在心包腔与腹壁交界处断裂,1根双极导线在Y形分叉处分成两个尖端电极时断裂。1次再次手术是由于起搏器召回。所有翻修手术均无并发症。
在我们研究所,幼儿的心外膜起搏临床效果令人满意,但也有相当数量的故障导致再次手术,主要是由于该患者群体的肌肉活动导致电极断裂。再次手术风险较低。