葡萄球菌蛋白A对补体增强的单纯疱疹病毒中和作用及病毒感染细胞免疫裂解的影响。
Effect of staphylococcal protein A on complement-potentiated neutralization of herpes simplex virus and immune lysis of virus-infected cells.
作者信息
Austin R M, Daniels C A
出版信息
Infect Immun. 1975 Oct;12(4):821-7. doi: 10.1128/iai.12.4.821-827.1975.
Abstract
Interaction of staphylococcal protein A (SPA) with human serum depressed the ability of such serum to neutralize herpes simpled virus (HSV)-antibody labialis. SPA-induced depression of serum-dependent virus neutralization appeared to be due to consumption of complement by SPA. In addition, SPA attached to antibody-treated, HSV-infected cells and inhibited complement mediated immune cytolysis. The amount of inhibition obtained depended upon the with the infected cells. The possible significance of SPA in the pathogenesis of viral disease complicated by secondary staphylococcal infection is discussed.
摘要
葡萄球菌蛋白A（SPA）与人血清的相互作用降低了该血清中和唇疱疹病毒（HSV）抗体的能力。SPA诱导的血清依赖性病毒中和作用降低似乎是由于SPA消耗了补体。此外，SPA附着于经抗体处理的HSV感染细胞上，并抑制补体介导的免疫细胞溶解。所获得的抑制量取决于与感染细胞的情况。本文讨论了SPA在继发葡萄球菌感染并发的病毒性疾病发病机制中的可能意义。
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