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本文引用的文献
1
IpaD localizes to the tip of the type III secretion system needle of Shigella flexneri.
Infect Immun. 2006 Aug;74(8):4391-400. doi: 10.1128/IAI.00440-06.
2
Characterization of the interaction of single tryptophan containing mutants of IpaC from Shigella flexneri with phospholipid membranes.
Biochemistry. 2006 Jan 17;45(2):626-36. doi: 10.1021/bi0512593.
3
Cholesterol binding by the bacterial type III translocon is essential for virulence effector delivery into mammalian cells.
Mol Microbiol. 2005 May;56(3):590-603. doi: 10.1111/j.1365-2958.2005.04568.x.
4
Optimization of virulence functions through glucosylation of Shigella LPS.
Science. 2005 Feb 25;307(5713):1313-7. doi: 10.1126/science.1108472.
5
Rafts can trigger contact-mediated secretion of bacterial effectors via a lipid-based mechanism.
J Biol Chem. 2004 Nov 12;279(46):47792-8. doi: 10.1074/jbc.M406824200. Epub 2004 Sep 10.
6
Type III secretion systems and bacterial flagella: insights into their function from structural similarities.
Proc Natl Acad Sci U S A. 2003 Mar 18;100(6):3027-30. doi: 10.1073/pnas.0535335100. Epub 2003 Mar 11.
7
Initial steps of Shigella infection depend on the cholesterol/sphingolipid raft-mediated CD44-IpaB interaction.
EMBO J. 2002 Sep 2;21(17):4449-57. doi: 10.1093/emboj/cdf457.
8
Structure and composition of the Shigella flexneri "needle complex", a part of its type III secreton.
Mol Microbiol. 2001 Feb;39(3):652-63. doi: 10.1046/j.1365-2958.2001.02200.x.
9
The tripartite type III secreton of Shigella flexneri inserts IpaB and IpaC into host membranes.
J Cell Biol. 1999 Nov 1;147(3):683-93. doi: 10.1083/jcb.147.3.683.
10
Secretion of Ipa proteins by Shigella flexneri: inducer molecules and kinetics of activation.
Infect Immun. 1997 Oct;65(10):4005-10. doi: 10.1128/iai.65.10.4005-4010.1997.
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