Enhanced fibrinolysis in fingerstick blood samples: a possible role for matrix metalloproteinase-9.
作者信息
Vanjani Rachna, Park Melissa, Holt Brittany, Cullinane Ann, Merryman Paula, Horne McDonald
机构信息
Hematology Service, Department of Laboratory Medicine, W. G. Magnuson Clinical Center, National Institutes of Health, Bethesda, MD 20892, USA.
出版信息
Thromb Res. 2007;120(5):773-8. doi: 10.1016/j.thromres.2006.12.018. Epub 2007 Apr 3.
Abstract
摘要
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本文引用的文献
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Effect of tranilast on matrix metalloproteinase production from neutrophils in-vitro.曲尼司特对中性粒细胞体外产生基质金属蛋白酶的影响。
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Plasminogen interaction with platelets: the importance of carboxyterminal lysines.纤溶酶原与血小板的相互作用：羧基末端赖氨酸的重要性。
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Molecular mechanisms of fibrinolysis.纤维蛋白溶解的分子机制
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Inducible expression of tissue inhibitor of metalloproteinases-resistant matrix metalloproteinase-9 on the cell surface of neutrophils.中性粒细胞表面金属蛋白酶组织抑制剂抗性基质金属蛋白酶-9的可诱导表达
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Matrix metalloproteinases and cellular fibrinolytic activity.
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Inactivation of plasminogen activator inhibitor-1 by specific proteolysis with stromelysin-1 (MMP-3).
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Characterization of stromelysin 1 (MMP-3), matrilysin (MMP-7), and membrane type 1 matrix metalloproteinase (MT1-MMP) derived fibrin(ogen) fragments D-dimer and D-like monomer: NH2-terminal sequences of late-stage digest fragments.基质溶解素1（MMP-3）、基质溶素（MMP-7）和膜型1基质金属蛋白酶（MT1-MMP）衍生的纤维蛋白（原）片段D-二聚体和D样单体的表征：晚期消化片段的氨基末端序列
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