Hamdan Abdul-Latif, Sabra Omar, Rameh Charbel, El-Khatib Mohamad
Department of Otolaryngology-Head & Neck Surgery, American University of Beirut.
Middle East J Anaesthesiol. 2007 Feb;19(1):5-13.
Voice production is a complex process that involves more than one system, yet most causes of dysphonia are attributed to disturbances in the laryngeal structures and little attention is paid to extralaryngeal factors. Persistent dysphonia after general anesthesia is a challenge to both anesthesiologists and otolaryngologists. The etiology is often multivariable and necessitates a team approach for proper diagnosis. Laryngeal symptoms are subdivided into phonatory disturbances and airway related complaints. When they become persistent for more than 72 hours or are coupled with airway symptoms such as hemoptysis, stridor, dyspnea or aspiration, the anesthesiologist should suspect injury to the vocal folds or cricoarytenoid joints. Here-below, the laryngeal manifestations of endotracheal intubation and the pathophysiology of vocal fold scarring are discussed.
发声是一个复杂的过程,涉及多个系统,但大多数发音障碍的原因都归因于喉部结构的紊乱,而很少关注喉外因素。全身麻醉后持续存在的发音障碍对麻醉医生和耳鼻喉科医生来说都是一个挑战。其病因通常是多因素的,需要团队协作进行正确诊断。喉部症状可细分为发声障碍和气道相关症状。当这些症状持续超过72小时或伴有咯血、喘鸣、呼吸困难或误吸等气道症状时,麻醉医生应怀疑声带或环杓关节受损。以下将讨论气管插管的喉部表现以及声带瘢痕形成的病理生理学。