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前沿:在缺乏外源性转化生长因子-β的情况下,调节性T细胞诱导CD4+CD25-Foxp3- T细胞或自身诱导成为Th17细胞。

Cutting edge: regulatory T cells induce CD4+CD25-Foxp3- T cells or are self-induced to become Th17 cells in the absence of exogenous TGF-beta.

作者信息

Xu Lili, Kitani Atsushi, Fuss Ivan, Strober Warren

机构信息

Mucosal Immunity Section, Laboratory of Host Defenses, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, MD 20892, USA.

出版信息

J Immunol. 2007 Jun 1;178(11):6725-9. doi: 10.4049/jimmunol.178.11.6725.

Abstract

Recent studies have shown that TGF-beta together with IL-6 induce the differentiation of IL-17-producing T cells (Th17) T cells. We therefore examined whether CD4(+)CD25(+)Foxp3(+) regulatory T cells, i.e., cells previously shown to produce TGF-beta, serve as Th17 inducers. We found that upon activation purified CD25(+) T cells (or sorted GFP(+) T cells obtained from Foxp3-GFP knockin mice) produce high amounts of soluble TGF-beta and when cultured with CD4(+)CD25(-)Foxp3(-) T cells in the presence of IL-6 induce the latter to differentiate into Th17 cells. Perhaps more importantly, upon activation, CD4(+)CD25(+)Foxp3(+)(GFP(+)) T cells themselves differentiate into Th17 cells in the presence of IL-6 (and in the absence of exogenous TGF-beta). These results indicate that CD4(+)CD25(+)Foxp3(+) regulatory T cells can function as inducers of Th17 cells and can differentiate into Th17 cells. They thus have important implications to our understanding of regulatory T cell function and their possible therapeutic use.

摘要

近期研究表明,转化生长因子β(TGF-β)与白细胞介素6(IL-6)共同诱导产生白细胞介素17的T细胞(Th17细胞)分化。因此,我们研究了CD4(+)CD25(+)Foxp3(+)调节性T细胞,即先前显示可产生TGF-β的细胞,是否作为Th17细胞诱导剂。我们发现,纯化的CD25(+)T细胞(或从Foxp3-GFP基因敲入小鼠获得的分选GFP(+)T细胞)激活后会产生大量可溶性TGF-β,并且在IL-6存在的情况下与CD4(+)CD25(-)Foxp3(-)T细胞共培养时,会诱导后者分化为Th17细胞。也许更重要的是,激活后,CD4(+)CD25(+)Foxp3(+)(GFP(+))T细胞自身在IL-6存在的情况下(且无外源性TGF-β)会分化为Th17细胞。这些结果表明,CD4(+)CD25(+)Foxp3(+)调节性T细胞可作为Th17细胞的诱导剂,并可分化为Th17细胞。因此,它们对于我们理解调节性T细胞功能及其可能的治疗用途具有重要意义。

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