血压控制与肾上腺素能过度激活的治疗调节 - Suppr

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超能文献

Blood pressure control and therapeutic modulation of the adrenergic overdrive.

作者信息

Grassi Guido, Trevano Fosca Quarti

机构信息

Clinica Medica, Università Milano-Bicocca, Ospedale San Gerardo, Via Pergolesi 33, 20052 Monza (Milan), Italy.

出版信息

Curr Hypertens Rep. 2007 Jun;9(3):167-9. doi: 10.1007/s11906-007-0029-z.

DOI:10.1007/s11906-007-0029-z

PMID:17519119

Abstract

摘要

相似文献

Blood pressure control and therapeutic modulation of the adrenergic overdrive.血压控制与肾上腺素能过度激活的治疗调节

Curr Hypertens Rep. 2007 Jun;9(3):167-9. doi: 10.1007/s11906-007-0029-z.

Antihypertensive treatment and sympathetic excitation.抗高血压治疗与交感神经兴奋

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Doxazosin suppresses the morning increase in blood pressure and sympathetic nervous activity in patients with essential hypertension.多沙唑嗪可抑制原发性高血压患者早晨血压升高及交感神经活动增强。

Hypertens Res. 1997 Sep;20(3):149-56. doi: 10.1291/hypres.20.149.

[Hypertension and the sympathetic nervous system - recent developments in research and treatment].[高血压与交感神经系统——研究与治疗的最新进展]

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[Effect of sympathetic nervous system on circadian rhythm of blood pressure].[交感神经系统对血压昼夜节律的影响]

Nihon Rinsho. 2014 Aug;72(8):1374-8.

Point: Chronic activation of the sympathetic nervous system is the dominant contributor to systemic hypertension.观点：交感神经系统的慢性激活是全身性高血压的主要促成因素。

J Appl Physiol (1985). 2010 Dec;109(6):1996-8; discussion 2016. doi: 10.1152/japplphysiol.00182.2010. Epub 2010 Feb 25.

Rebuttal from Esler, Lambert, and Schlaich.埃斯勒、兰伯特和施莱希的反驳。

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Comments on Point:Counterpoint: The dominant contributor to systemic hypertension: Chronic activation of the sympathetic nervous system vs. Activation of the intrarenal renin-angiotensin system. Sympathetic nervous system, plasma volume, and hypertension.关于“针锋相对：系统性高血压的主要促成因素：交感神经系统的慢性激活与肾内肾素 - 血管紧张素系统的激活”的评论。交感神经系统、血容量与高血压。

J Appl Physiol (1985). 2010 Dec;109(6):2003. doi: 10.1152/japplphysiol.01160.2010.

The importance of sleep blood pressure.睡眠血压的重要性。

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本文引用的文献

Counteracting the sympathetic nervous system in essential hypertension.

Curr Opin Nephrol Hypertens. 2004 Sep;13(5):513-9. doi: 10.1097/00041552-200409000-00006.

Morning blood pressure surge and hypertensive cerebrovascular disease: role of the alpha adrenergic sympathetic nervous system.晨起血压激增与高血压性脑血管疾病：α肾上腺素能交感神经系统的作用

Am J Hypertens. 2004 Aug;17(8):668-75. doi: 10.1016/j.amjhyper.2004.04.001.

Effect of morning and bedtime dosing with cilnidipine on blood pressure, heart rate, and sympathetic nervous activity in essential hypertensive patients.西尼地平早晚给药对原发性高血压患者血压、心率及交感神经活动的影响。

J Cardiovasc Pharmacol. 2004 Jan;43(1):68-73. doi: 10.1097/00005344-200401000-00011.

Sympathetic deactivation as a goal of nonpharmacologic and pharmacologic antihypertensive treatment: rationale and options.交感神经失活作为非药物和药物降压治疗的目标：理论依据与选择

Curr Hypertens Rep. 2003 Aug;5(4):277-80. doi: 10.1007/s11906-003-0033-x.

2003 European Society of Hypertension-European Society of Cardiology guidelines for the management of arterial hypertension.2003年欧洲高血压学会-欧洲心脏病学会动脉高血压管理指南

J Hypertens. 2003 Jun;21(6):1011-53. doi: 10.1097/00004872-200306000-00001.

Morning surge in blood pressure as a predictor of silent and clinical cerebrovascular disease in elderly hypertensives: a prospective study.老年高血压患者晨峰血压作为隐匿性和临床脑血管疾病预测指标的前瞻性研究

Circulation. 2003 Mar 18;107(10):1401-6. doi: 10.1161/01.cir.0000056521.67546.aa.

Short-versus long-term effects of different dihydropyridines on sympathetic and baroreflex function in hypertension.不同二氢吡啶对高血压患者交感神经和压力反射功能的短期与长期影响

Hypertension. 2003 Mar;41(3):558-62. doi: 10.1161/01.HYP.0000058003.27729.5A. Epub 2003 Feb 17.

Systolic and diastolic blood pressure control in antihypertensive drug trials.

J Hypertens. 2002 Aug;20(8):1461-4. doi: 10.1097/00004872-200208000-00001.

Sympathetic neural mechanisms in white-coat hypertension.白大衣高血压中的交感神经机制。

J Am Coll Cardiol. 2002 Jul 3;40(1):126-32. doi: 10.1016/s0735-1097(02)01931-9.

Systolic vs diastolic blood pressure control in the hypertensive patients of the PAMELA population. Pressioni Arteriose Monitorate E Loro Associazioni.PAMELA人群中高血压患者的收缩压与舒张压控制。动脉血压监测及其关联研究

Arch Intern Med. 2002 Mar 11;162(5):582-6. doi: 10.1001/archinte.162.5.582.

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