Studying hepatitis C virus: making the best of a bad virus.
作者信息
Tellinghuisen Timothy L, Evans Matthew J, von Hahn Thomas, You Shihyun, Rice Charles M
机构信息
The Scripps Research Institute, 5353 Parkside Drive, RF-2, Jupiter, FL 33458, USA.
出版信息
J Virol. 2007 Sep;81(17):8853-67. doi: 10.1128/JVI.00753-07. Epub 2007 May 23.
Abstract
摘要
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本文引用的文献
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Phosphorylation of hepatitis C virus NS5A nonstructural protein: a new paradigm for phosphorylation-dependent viral RNA replication?丙型肝炎病毒NS5A非结构蛋白的磷酸化：磷酸化依赖性病毒RNA复制的新范例？
Virology. 2007 Jul 20;364(1):1-9. doi: 10.1016/j.virol.2007.01.042. Epub 2007 Apr 2.
2
Hepatitis C virus production by human hepatocytes dependent on assembly and secretion of very low-density lipoproteins.人肝细胞产生丙型肝炎病毒依赖于极低密度脂蛋白的组装和分泌。
Proc Natl Acad Sci U S A. 2007 Apr 3;104(14):5848-53. doi: 10.1073/pnas.0700760104. Epub 2007 Mar 21.
3
Claudin-1 is a hepatitis C virus co-receptor required for a late step in entry.Claudin-1是丙型肝炎病毒进入后期阶段所需的共受体。
Nature. 2007 Apr 12;446(7137):801-5. doi: 10.1038/nature05654. Epub 2007 Feb 25.
4
Analysis of hepatitis C virus superinfection exclusion by using novel fluorochrome gene-tagged viral genomes.利用新型荧光染料基因标记病毒基因组分析丙型肝炎病毒重叠感染的排除情况。
J Virol. 2007 May;81(9):4591-603. doi: 10.1128/JVI.02144-06. Epub 2007 Feb 14.
5
Participation of rab5, an early endosome protein, in hepatitis C virus RNA replication machinery.早期内体蛋白rab5参与丙型肝炎病毒RNA复制机制。
J Virol. 2007 May;81(9):4551-63. doi: 10.1128/JVI.01366-06. Epub 2007 Feb 14.
6
Production of infectious hepatitis C virus of various genotypes in cell cultures.在细胞培养物中产生多种基因型的传染性丙型肝炎病毒。
J Virol. 2007 May;81(9):4405-11. doi: 10.1128/JVI.02334-06. Epub 2007 Feb 14.
7
Evidence for a functional RNA element in the hepatitis C virus core gene.丙型肝炎病毒核心基因中功能性RNA元件的证据。
Proc Natl Acad Sci U S A. 2007 Feb 20;104(8):2879-84. doi: 10.1073/pnas.0611267104. Epub 2007 Feb 13.
8
Superinfection exclusion in cells infected with hepatitis C virus.丙型肝炎病毒感染细胞中的超感染排除
J Virol. 2007 Apr;81(8):3693-703. doi: 10.1128/JVI.01748-06. Epub 2007 Feb 7.
9
Scavenger receptor BI and BII expression levels modulate hepatitis C virus infectivity.清道夫受体BI和BII的表达水平调节丙型肝炎病毒的感染性。
J Virol. 2007 Apr;81(7):3162-9. doi: 10.1128/JVI.02356-06. Epub 2007 Jan 10.
10
Hepatitis C virus NS5A is a direct substrate of casein kinase I-alpha, a cellular kinase identified by inhibitor affinity chromatography using specific NS5A hyperphosphorylation inhibitors.丙型肝炎病毒NS5A是酪蛋白激酶I-α的直接底物，酪蛋白激酶I-α是一种细胞激酶，通过使用特异性NS5A过度磷酸化抑制剂的抑制剂亲和色谱法鉴定。
J Biol Chem. 2007 Feb 23;282(8):5536-44. doi: 10.1074/jbc.M610486200. Epub 2006 Dec 13.
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