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本文引用的文献

1
TDP-43-positive white matter pathology in frontotemporal lobar degeneration with ubiquitin-positive inclusions.伴有泛素阳性包涵体的额颞叶变性中的TDP - 43阳性白质病变
J Neuropathol Exp Neurol. 2007 Mar;66(3):177-83. doi: 10.1097/01.jnen.0000248554.45456.58.
2
Neuropathologic heterogeneity in HDDD1: a familial frontotemporal lobar degeneration with ubiquitin-positive inclusions and progranulin mutation.HDDD1中的神经病理学异质性:一种伴有泛素阳性包涵体和原颗粒蛋白突变的家族性额颞叶痴呆
Alzheimer Dis Assoc Disord. 2007 Jan-Mar;21(1):1-7. doi: 10.1097/WAD.0b013e31803083f2.
3
TDP-43 in the ubiquitin pathology of frontotemporal dementia with VCP gene mutations.VCP基因突变的额颞叶痴呆泛素病理学中的TDP-43
J Neuropathol Exp Neurol. 2007 Feb;66(2):152-7. doi: 10.1097/nen.0b013e31803020b9.
4
Ubiquitinated pathological lesions in frontotemporal lobar degeneration contain the TAR DNA-binding protein, TDP-43.额颞叶变性中的泛素化病理病变包含TAR DNA结合蛋白TDP-43。
Acta Neuropathol. 2007 May;113(5):521-33. doi: 10.1007/s00401-006-0189-y. Epub 2007 Jan 12.
5
Analysis of IFT74 as a candidate gene for chromosome 9p-linked ALS-FTD.IFT74作为9号染色体连锁的肌萎缩侧索硬化症-额颞叶痴呆候选基因的分析。
BMC Neurol. 2006 Dec 13;6:44. doi: 10.1186/1471-2377-6-44.
6
TDP-43 is a component of ubiquitin-positive tau-negative inclusions in frontotemporal lobar degeneration and amyotrophic lateral sclerosis.TDP-43是额颞叶痴呆和肌萎缩侧索硬化中泛素阳性、tau蛋白阴性包涵体的一个组成部分。
Biochem Biophys Res Commun. 2006 Dec 22;351(3):602-11. doi: 10.1016/j.bbrc.2006.10.093. Epub 2006 Oct 30.
7
The neuropathology of frontotemporal lobar degeneration caused by mutations in the progranulin gene.由原颗粒蛋白基因突变引起的额颞叶变性的神经病理学
Brain. 2006 Nov;129(Pt 11):3081-90. doi: 10.1093/brain/awl271.
8
Ubiquitinated TDP-43 in frontotemporal lobar degeneration and amyotrophic lateral sclerosis.额颞叶痴呆和肌萎缩侧索硬化症中泛素化的TDP-43
Science. 2006 Oct 6;314(5796):130-3. doi: 10.1126/science.1134108.
9
Pathological heterogeneity of frontotemporal lobar degeneration with ubiquitin-positive inclusions delineated by ubiquitin immunohistochemistry and novel monoclonal antibodies.通过泛素免疫组织化学和新型单克隆抗体描绘的伴有泛素阳性包涵体的额颞叶变性的病理异质性
Am J Pathol. 2006 Oct;169(4):1343-52. doi: 10.2353/ajpath.2006.060438.
10
HDDD2 is a familial frontotemporal lobar degeneration with ubiquitin-positive, tau-negative inclusions caused by a missense mutation in the signal peptide of progranulin.HDDD2是一种家族性额颞叶痴呆,由颗粒蛋白前体信号肽中的错义突变引起,具有泛素阳性、tau阴性包涵体。
Ann Neurol. 2006 Sep;60(3):314-22. doi: 10.1002/ana.20963.

伴有泛素包涵体的家族性和散发性额颞叶痴呆中的TDP-43

TDP-43 in familial and sporadic frontotemporal lobar degeneration with ubiquitin inclusions.

作者信息

Cairns Nigel J, Neumann Manuela, Bigio Eileen H, Holm Ida E, Troost Dirk, Hatanpaa Kimmo J, Foong Chan, White Charles L, Schneider Julie A, Kretzschmar Hans A, Carter Deborah, Taylor-Reinwald Lisa, Paulsmeyer Katherine, Strider Jeffrey, Gitcho Michael, Goate Alison M, Morris John C, Mishra Manjari, Kwong Linda K, Stieber Anna, Xu Yan, Forman Mark S, Trojanowski John Q, Lee Virginia M-Y, Mackenzie Ian R A

机构信息

MRCPath, Department of Pathology and Immunology, Washington University School of Medicine, Campus Box 8118, St Louis, MO 63110, USA.

出版信息

Am J Pathol. 2007 Jul;171(1):227-40. doi: 10.2353/ajpath.2007.070182.

DOI:10.2353/ajpath.2007.070182
PMID:17591968
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC1941578/
Abstract

TAR DNA-binding protein 43 (TDP-43) is a major pathological protein of sporadic and familial frontotemporal lobar degeneration with ubiquitin-positive, tau-negative inclusions (FTLD-U) with or without motor neuron disease (MND). Thus, TDP-43 defines a novel class of neurodegenerative diseases called TDP-43 proteinopathies. We performed ubiquitin and TDP-43 immunohistochemistry on 193 cases of familial and sporadic FTLD with or without MND. On selected cases, immunoelectron microscopy and biochemistry were performed. Clinically defined frontotemporal dementias (FTDs) included four groups: 1) familial FTD with mutations in progranulin (n = 36), valosin-containing protein (n = 5), charged multivesicular body protein 2B (n = 4), and linked to chromosome 9p (n = 7); 2) familial cases of FTD with unknown gene association (n = 29); 3) sporadic FTD (n = 72); and 4) familial and sporadic FTD with MND (n = 40). Our studies confirm that the spectrum of TDP-43 proteinopathies includes most cases of sporadic and familial FTLD-U with and without MND and expand this disease spectrum to include reported families with FTD linked to chromosome 9p but not FTD with charged multivesicular body protein 2B mutations. Thus, despite significant clinical, genetic, and neuropathological heterogeneity of FTLD-U, TDP-43 is a common pathological substrate underlying a large subset of these disorders, thereby implicating TDP-43 in novel and unifying mechanisms of FTLD pathogenesis.

摘要

TAR DNA结合蛋白43(TDP - 43)是散发性和家族性额颞叶变性伴泛素阳性、tau阴性包涵体(FTLD - U)(伴或不伴运动神经元病(MND))的主要病理蛋白。因此,TDP - 43定义了一类新的神经退行性疾病,称为TDP - 43蛋白病。我们对193例伴或不伴MND的家族性和散发性FTLD病例进行了泛素和TDP - 43免疫组织化学检测。对部分病例进行了免疫电子显微镜检查和生物化学分析。临床定义的额颞叶痴呆(FTD)包括四组:1)伴有颗粒前体蛋白突变的家族性FTD(n = 36)、含缬酪肽蛋白突变(n = 5)、多囊泡体蛋白2B突变(n = 4)以及与9号染色体p臂连锁(n = 7);2)基因关联不明的家族性FTD病例(n = 29);3)散发性FTD(n = 72);4)伴MND的家族性和散发性FTD(n = 40)。我们的研究证实,TDP - 43蛋白病谱包括大多数伴或不伴MND的散发性和家族性FTLD - U病例,并将该疾病谱扩展至包括报道的与9号染色体p臂连锁的FTD家族,但不包括有多囊泡体蛋白2B突变的FTD。因此,尽管FTLD - U在临床、遗传和神经病理学上存在显著异质性,但TDP - 43是这些疾病很大一部分的共同病理基础,从而提示TDP - 43参与了FTLD发病机制中的新的统一机制。