Todnem K, Vaernes R, Kambestad B K
Norwegian Underwater Technology Centre, Bergen.
Aviat Space Environ Med. 1991 Oct;62(10):982-5.
Visual evoked potentials (VEP) were examined in 14 divers during dives to 360 metres of seawater (msw). All latencies increased significantly with depth. VEP and brain stem auditory evoked potentials (BAEP) were similarly examined in 18 divers before and after these dives. N75 was significantly increased after compared to before the dive, while there was no significant difference in the P100 and N145 latencies. BAEP I-V latency was significantly decreased after the dives. VEP and BAEP were examined in 156 divers and 99 controls. There was no significant difference in VEP. BAEP I-III interpeak latency was significantly increased for the divers, but with no significant changes in I-V and III-V latencies. VEP and BAEP were examined in 26 divers after treatment for neurological decompression sickness. There was no significant difference compared to the control group. The conclusions are that VEP and BAEP change transitorily with influence of hyperbaric pressure, but do not measure major permanent disturbances in the divers' visual and auditory pathways.
对14名潜水员在潜入360米海水深度时进行了视觉诱发电位(VEP)检查。所有潜伏期均随深度显著增加。在这些潜水前后,对18名潜水员同样进行了VEP和脑干听觉诱发电位(BAEP)检查。与潜水前相比,潜水后N75显著增加,而P100和N145潜伏期无显著差异。潜水后BAEP I-V潜伏期显著缩短。对156名潜水员和99名对照组人员进行了VEP和BAEP检查。VEP无显著差异。潜水员的BAEP I-III峰间潜伏期显著延长,但I-V和III-V潜伏期无显著变化。对26名接受神经型减压病治疗后的潜水员进行了VEP和BAEP检查。与对照组相比无显著差异。结论是,VEP和BAEP会因高压的影响而短暂变化,但无法检测到潜水员视觉和听觉通路中的主要永久性障碍。