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Molecular aspects of neuronal voltage-dependent K+ channels.

作者信息

Rehm H

机构信息

Pharmakologisches Institut, Universität Zürich, Switzerland.

出版信息

Eur J Biochem. 1991 Dec 18;202(3):701-13. doi: 10.1111/j.1432-1033.1991.tb16425.x.

DOI:10.1111/j.1432-1033.1991.tb16425.x
PMID:1765087
Abstract
摘要

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Molecular aspects of neuronal voltage-dependent K+ channels.
Eur J Biochem. 1991 Dec 18;202(3):701-13. doi: 10.1111/j.1432-1033.1991.tb16425.x.
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Mutations affecting TEA blockade and ion permeation in voltage-activated K+ channels.影响电压激活钾通道中TEA阻断和离子通透的突变。
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A neuronal beta subunit (KCNMB4) makes the large conductance, voltage- and Ca2+-activated K+ channel resistant to charybdotoxin and iberiotoxin.一种神经元β亚基(KCNMB4)使大电导、电压和钙离子激活的钾通道对蝎毒素和iberiotoxin具有抗性。
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Different types of K+ channel current are generated by different levels of a single mRNA.不同类型的钾离子通道电流由单个信使核糖核酸的不同水平产生。
EMBO J. 1992 Jul;11(7):2465-71. doi: 10.1002/j.1460-2075.1992.tb05311.x.
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