Lachman M, Brzek A, Mellan J, Hampl R, Starka L, Motlik K
Unit of Sexology, OUNZ, Charles University, Prague, Czechoslovakia.
Exp Clin Endocrinol. 1991;98(2):171-4. doi: 10.1055/s-0029-1211114.
The case of recidivous sexual offender with genetically caused mental retardation and primary hypogonadism (Klinefelter's syndrome with karyotype 48, XXXY) is described. He was examined after sadistic abuse of a boy aged 13 that he had committed 19 years after performed testicular pulpectomy. Plasmatic level of testosterone was found 4x higher than mean level in men after orchidectomy. Histological examination of residual scrotal tissues proved that the source of androgens were hyperplastic nodules of extratesticular Leydig cells.
本文描述了一例患有遗传性智力障碍和原发性性腺功能减退（核型为48, XXXY的克兰费尔特综合征）的复发性性犯罪者的病例。他在对一名13岁男孩实施虐待行为后接受检查，该虐待行为发生在他进行睾丸摘除术后19年。发现其血浆睾酮水平比睾丸切除术后男性的平均水平高4倍。阴囊残余组织的组织学检查证明，雄激素的来源是睾丸外莱迪希细胞的增生性结节。
