Revealing the cellular basis of heart failure.
作者信息
Bridge John H B, Savio Eleonora
机构信息
Nora Eccles Harrison CardiovascularResearch and Training Institute, Division of Cardiology, University of Utah, Salt Lake City, Utah, USA.
出版信息
Biophys J. 2007 Dec 1;93(11):3731-2. doi: 10.1529/biophysj.107.116541. Epub 2007 Sep 7.
Abstract
摘要
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Enhanced ryanodine receptor-mediated calcium leak determines reduced sarcoplasmic reticulum calcium content in chronic canine heart failure.增强的兰尼碱受体介导的钙泄漏决定了慢性犬心力衰竭时肌浆网钙含量的降低。
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History of heart failure (including hypertension).心力衰竭病史（包括高血压）。
Z Kardiol. 2002;91 Suppl 4:60-3. doi: 10.1007/s00392-002-1410-1.
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Decreased sarcoplasmic reticulum calcium content is responsible for defective excitation-contraction coupling in canine heart failure.肌浆网钙含量降低是犬心力衰竭时兴奋 - 收缩偶联缺陷的原因。
Circulation. 2001 Mar 20;103(11):1577-84. doi: 10.1161/01.cir.103.11.1577.
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PKA phosphorylation dissociates FKBP12.6 from the calcium release channel (ryanodine receptor): defective regulation in failing hearts.蛋白激酶A磷酸化使FKBP12.6从钙释放通道（雷诺丁受体）上解离：在衰竭心脏中调节功能存在缺陷。
Cell. 2000 May 12;101(4):365-76. doi: 10.1016/s0092-8674(00)80847-8.
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ABC of heart failure. History and epidemiology.心力衰竭基础。病史与流行病学。
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