Chen Z, Qing D, Zheng Y, Xie B
Department of Physiology.
Hua Xi Yi Ke Da Xue Xue Bao. 1991 Jun;22(2):144-7.
Experiments were performed on 18 rabbits, bivagotomized, gallamine triethiodide immobilized and artificially ventilated. The effects of electrical stimulation of "renzhong" (Du 26) area and NPBM, separately and simultaneously, on arterial blood pressure and heart rate were observed before and after electrolytic or chemical lesion of NPBM. Electrical stimulation of "renzhong" area or NPBM, separately could elicit an increase in arterial blood pressure; when both "renzhong" and NPBM were stimulated, the pressor response to NPBM stimulation could affect that of "renzhong" area's. But after electrolytic or chemical lesion of NPBM, stimulation of NPBM could no longer elicit pressor response. On the other hand, the pressor response induced by electrical stimulation of "renzhong" area decreased obviously, after lesion of NPBM or transection of brain stem between NPBM and Nucleus trigemini principalis. These results suggest that NPBM might take part in the pressor response of "renzhong" area stimulation.
对18只经双迷走神经切断、三碘季铵酚制动并进行人工通气的家兔进行了实验。在对延髓头端腹外侧网状结构（NPBM）进行电解或化学损伤前后，分别观察了电刺激“人中”（督脉26）区和NPBM以及同时刺激二者对动脉血压和心率的影响。单独电刺激“人中”区或NPBM均可引起动脉血压升高；当同时刺激“人中”区和NPBM时，对NPBM刺激的升压反应会影响“人中”区的升压反应。但在对NPBM进行电解或化学损伤后，刺激NPBM不再引起升压反应。另一方面，在NPBM损伤或在NPBM与三叉神经主核之间横断脑干后，电刺激“人中”区所诱发的升压反应明显减弱。这些结果提示，NPBM可能参与了刺激“人中”区的升压反应。
