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Involvement of SH-groups during interaction of diazoxide with the inner membrane of rat heart mitochondria.

作者信息

Korotkov S M, Nesterov V P, Emel'yanova L V, Ryabchikov N N

机构信息

Sechenov Institute of Evolutionary Physiology and Biochemistry, Russian Academy of Sciences, pr Morisa Toreza 44, St. Petersburg, 194223, Russia.

出版信息

Dokl Biochem Biophys. 2007 Jul-Aug;415:206-10. doi: 10.1134/s1607672907040126.

DOI:10.1134/s1607672907040126
PMID:17933337
Abstract
摘要

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Involvement of SH-groups during interaction of diazoxide with the inner membrane of rat heart mitochondria.二氮嗪与大鼠心脏线粒体内膜相互作用过程中SH基团的参与情况。
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2
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[Effects of diazoxide on the mitochondrial membrane potential and ROS generation in rat uterus cells].[二氮嗪对大鼠子宫细胞线粒体膜电位及活性氧生成的影响]
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Diazoxide-induced cardioprotection via DeltaPsim loss depending on timing of application.二氮嗪通过依赖于应用时间的线粒体膜电位丧失诱导心脏保护作用。
Life Sci. 2006 Oct 12;79(20):1906-12. doi: 10.1016/j.lfs.2006.06.012. Epub 2006 Jun 15.
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Opening of mitochondrial K(ATP) channels triggers cardioprotection. Are reactive oxygen species involved?线粒体ATP敏感性钾通道的开放触发心脏保护作用。活性氧是否参与其中？
Circ Res. 2001 Apr 27;88(8):750-2. doi: 10.1161/hh0801.090537.
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The KATP channel opener diazoxide protects cardiac myocytes during metabolic inhibition without causing mitochondrial depolarization or flavoprotein oxidation.KATP通道开放剂二氮嗪在代谢抑制期间可保护心肌细胞，且不会引起线粒体去极化或黄素蛋白氧化。
Br J Pharmacol. 2001 Oct;134(3):535-42. doi: 10.1038/sj.bjp.0704289.
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Study of the mechanism of action of diazoxide on rat heart mitochondria under calcium loading.钙负荷下二氮嗪对大鼠心脏线粒体作用机制的研究。
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Diazoxide and pinacidil uncouple pyruvate-malate-induced mitochondrial respiration.二氮嗪和平尼地尔可使丙酮酸-苹果酸诱导的线粒体呼吸解偶联。
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Myocardial preconditioning against ischemia-reperfusion injury is abolished in Zucker obese rats with insulin resistance.在患有胰岛素抵抗的Zucker肥胖大鼠中，针对缺血再灌注损伤的心肌预处理作用消失。
Am J Physiol Regul Integr Comp Physiol. 2007 Feb;292(2):R920-6. doi: 10.1152/ajpregu.00520.2006. Epub 2006 Sep 28.

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Effects of Tl(+) on ion permeability, membrane potential and respiration of isolated rat liver mitochondria.铊离子（Tl⁺）对离体大鼠肝线粒体离子通透性、膜电位及呼吸作用的影响。
J Bioenerg Biomembr. 2009 Jun;41(3):277-87. doi: 10.1007/s10863-009-9225-7. Epub 2009 Jul 22.
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本文引用的文献

1
Study of the mechanism of action of diazoxide on rat heart mitochondria under calcium loading.钙负荷下二氮嗪对大鼠心脏线粒体作用机制的研究。
Dokl Biochem Biophys. 2006 May-Jun;408:133-7. doi: 10.1134/s1607672906030070.
2
Combined modulation of the mitochondrial ATP-dependent potassium channel and the permeability transition pore causes prolongation of the biphasic calcium dynamics.线粒体ATP依赖性钾通道和通透性转换孔的联合调节导致双相钙动力学延长。
Cell Calcium. 2006 May;39(5):387-400. doi: 10.1016/j.ceca.2006.01.001. Epub 2006 Mar 2.
3
Mitochondrial K(ATP) channels in cell survival and death.
巯基对大鼠心脏线粒体呼吸及肿胀影响的研究
Dokl Biochem Biophys. 2008 Jul-Aug;421:171-5. doi: 10.1134/s1607672908040030.
细胞存活与死亡中的线粒体ATP敏感性钾通道
J Mol Cell Cardiol. 2005 Jul;39(1):7-16. doi: 10.1016/j.yjmcc.2004.12.003. Epub 2005 Feb 19.
4
K(ATP) channels and preconditioning: a re-examination of the role of mitochondrial K(ATP) channels and an overview of alternative mechanisms.钾离子通道与预处理：对线粒体钾离子通道作用的重新审视及其他机制概述
J Mol Cell Cardiol. 2005 Jul;39(1):17-50. doi: 10.1016/j.yjmcc.2005.04.002.
5
Calcium-activated potassium channel triggers cardioprotection of ischemic preconditioning.钙激活钾通道触发缺血预处理的心脏保护作用。
J Pharmacol Exp Ther. 2005 Feb;312(2):644-50. doi: 10.1124/jpet.104.074476. Epub 2004 Sep 2.
6
Potassium channel openers are uncoupling protonophores: implication in cardioprotection.钾通道开放剂是解偶联质子载体：对心脏保护的意义。
FEBS Lett. 2004 Jun 18;568(1-3):167-70. doi: 10.1016/j.febslet.2004.05.031.
7
Alterations of the bioenergetics systems of the cell in acute and chronic myocardial ischemia.急性和慢性心肌缺血时细胞生物能量系统的改变。
Mol Cell Biochem. 2004 Jan-Feb;256-257(1-2):157-66. doi: 10.1023/b:mcbi.0000009866.75225.e2.
8
Adenine nucleotide translocase mediates the K(ATP)-channel-openers-induced proton and potassium flux to the mitochondrial matrix.腺嘌呤核苷酸转位酶介导钾离子ATP通道开放剂诱导的质子和钾离子向线粒体基质的通量。
J Bioenerg Biomembr. 2003 Apr;35(2):141-8. doi: 10.1023/a:1023746103401.
9
On the effect of diazoxide on isolated mitochondria from rat myocardium.
Dokl Biochem Biophys. 2002 Nov-Dec;387:306-9. doi: 10.1023/a:1021788029457.
10
Role of critical thiol groups on the matrix surface of the adenine nucleotide translocase in the mechanism of the mitochondrial permeability transition pore.关键硫醇基团在腺嘌呤核苷酸转位酶基质表面在线粒体通透性转换孔机制中的作用
Biochem J. 2002 Oct 15;367(Pt 2):541-8. doi: 10.1042/BJ20011672.