Evans Gary W, Kim Pilyoung
Department of Design and Environmental Analysis, Cornell University, Ithaca, NY 14853-4401, USA.
Psychol Sci. 2007 Nov;18(11):953-7. doi: 10.1111/j.1467-9280.2007.02008.x.
A massive literature documents the inverse association between poverty or low socioeconomic status and health, but little is known about the mechanisms underlying this robust relation. We examined longitudinal relations between duration of poverty exposure since birth, cumulative risk exposure, and physiological stress in two hundred seven 13-year-olds. Chronic stress was assessed by basal blood pressure and overnight cortisol levels; stress regulation was assessed by cardiovascular reactivity to a standard acute stressor and recovery after exposure to this stressor. Cumulative risk exposure was measured by multiple physical (e.g., substandard housing) and social (e.g., family turmoil) risk factors. The greater the number of years spent living in poverty, the more elevated was overnight cortisol and the more dysregulated was the cardiovascular response (i.e., muted reactivity). Cardiovascular recovery was not affected by duration of poverty exposure. Unlike the duration of poverty exposure, concurrent poverty (i.e., during adolescence) did not affect these physiological stress outcomes. The effects of childhood poverty on stress dysregulation are largely explained by cumulative risk exposure accompanying childhood poverty.
大量文献记载了贫困或低社会经济地位与健康之间的负相关关系,但对于这种稳固关系背后的机制却知之甚少。我们研究了207名13岁青少年自出生以来的贫困暴露时长、累积风险暴露与生理应激之间的纵向关系。通过基础血压和夜间皮质醇水平评估慢性应激;通过对标准急性应激源的心血管反应性以及暴露于该应激源后的恢复情况评估应激调节。累积风险暴露通过多种身体(如住房条件差)和社会(如家庭动荡)风险因素来衡量。生活在贫困中的年数越多,夜间皮质醇水平越高,心血管反应失调越严重(即反应性减弱)。心血管恢复不受贫困暴露时长的影响。与贫困暴露时长不同,同时期贫困(即青春期期间)并未影响这些生理应激结果。童年贫困对应激失调的影响在很大程度上可由童年贫困伴随的累积风险暴露来解释。