Suzuki T
Department of Ophthalmology, Koshigaya Hospital, Dokkyo University School of Medicine, Japan.
Nippon Ganka Gakkai Zasshi. 1991 Sep;95(9):878-82.
Rebound nystagmus represents a disorder of ocular movement in patients with cerebellar lesions. It was reported, however, that it occurred in normal subjects in darkness. Jerk nystagmus, amplitude of 0.5-2 deg and frequency of less than 2Hz, was evoked after prolonged eccentric fixation and fatigue within about 5 to 15 seconds in 7 normal subjects of the experiment. The nystagmus increased depending on the eccentricity and duration of gaze holding. It also depended on the orbital position 10deg left, 0 deg, 10 deg right after eccentric gaze. It is suggested that rebound nystagmus resulted from the leaky neural integrator of the brainstem, and a shift of the null position in the direction of the eccentric gaze. These two components may cause different types of nystagmus by various combinations.
眼震反跳现象是小脑病变患者眼球运动的一种障碍。然而，据报道，在黑暗环境中的正常受试者身上也会出现这种现象。在该实验的7名正常受试者中，长时间偏中心注视和疲劳后，约5至15秒内会诱发幅度为0.5 - 2度、频率低于2Hz的急跳性眼震。眼震会随着注视偏心率和持续时间的增加而增强。它还取决于偏中心注视后眼眶位置（向左10度、0度、向右10度）。提示眼震反跳现象是由脑干神经积分器泄漏以及零位置向偏中心注视方向偏移所致。这两个因素可能通过各种组合导致不同类型的眼震。
