Trojan S, Kapitola J
Fyziologický ústav 1. lékarské fakulty Univerzity Karlovy, Praha.
Sb Lek. 1991 Jun;93(5-6):129-35.
The authors draw attention to the danger of the development of hyperaemia because of the possible influence of free oxygen radicals during the hypoxic period on brain cells. Using radioactive microparticles of 85Sr the authors assessed the cardiac minute volume and blood flow through the medulla oblongata, cerebellum, subcortical area and cerebral cortex in adult laboratory rats 20 hours after 2-minute ischaemia induced by positive radial acceleration of 10 G. The results indicate an increase of the local blood supply in the investigated parts of the brain, although lower than after 8-hour exposure to high altitude hypoxia at 7000 m.
作者提醒注意充血发展的危险，因为在缺氧期游离氧自由基可能对脑细胞产生影响。作者使用85Sr放射性微粒评估了成年实验大鼠在10G正向径向加速度诱导的2分钟缺血20小时后的心输出量以及通过延髓、小脑、皮层下区域和大脑皮层的血流量。结果表明，大脑被研究部位的局部血液供应增加，尽管低于在7000米高空缺氧8小时后的情况。
