Monroy Angelo, Behar Philomena, Brodsky Linda
Department of Otolaryngology, State University of New York at Buffalo, School of Medicine and Biomedical Sciences, NY 14202, United States.
Int J Pediatr Otorhinolaryngol. 2008 May;72(5):565-70. doi: 10.1016/j.ijporl.2008.01.008. Epub 2008 Mar 4.
Adenoid "re-growth" is a poorly understood phenomenon. While parents often express concerns regarding the potential for adenoid "re-growth", little information exists in the literature about its incidence and causation.
To establish the incidence and possible contributing factors leading to adenoid re-growth in children.
Retrospective case series review.
Tertiary care children's hospital.
The charts of 106 patients who underwent revision adenoidectomy between 1995 and 2006 were reviewed. Thirty-four patients were excluded because the primary adenoidectomy was performed elsewhere or initially only a partial adenoidectomy was performed. In the remaining 72 patients, demographic data, clinical presentation, associated medical conditions and findings at surgery were studied.
During the 11-year study period. 13,005 adenoidectomies or adenotonsillectomies were performed; 72/13,005 (0.55%) underwent revision adenoidectomy. The mean (+/-S.D.) age at presentation for primary adenoidectomy was 3.68+/-2.9 and 7.69+/-4.04 years for secondary ("revision") adenoidectomy with an average time interval of 4.3 years between surgeries. Age at initial adenoidectomy was not a significant factor in predicting revision adenoid surgery. 29/72 (40%) underwent a reflux work up including scintiscan with gastric emptying, 24h pH probe, or laryngoscopy. 28/29 (96%) were diagnosed with reflux. At least 15/72 (21%) were reported to have symptoms consistent with adenoid re-growth which were found to be caused by tubal tonsil hyperplasia.
Revision adenoidectomy rarely needs to be performed. Tubal tonsillar hyperplasia, as opposed to re-growth of residual adenoid tissue previously removed, accounts for some cases. Extraesophageal reflux is a possible cause in some and requires further study.
腺样体“再生长”是一种尚未被充分理解的现象。虽然家长们常常对腺样体“再生长”的可能性表示担忧,但文献中关于其发生率和病因的信息却很少。
确定儿童腺样体再生长的发生率及可能的相关因素。
回顾性病例系列研究。
三级护理儿童医院。
回顾了1995年至2006年间接受再次腺样体切除术的106例患者的病历。34例患者被排除,原因是初次腺样体切除术在其他地方进行或最初仅进行了部分腺样体切除术。在其余72例患者中,研究了人口统计学数据、临床表现、相关疾病状况及手术中的发现。
在11年的研究期间,共进行了13005例腺样体切除术或腺样体扁桃体切除术;72/13005(0.55%)接受了再次腺样体切除术。初次腺样体切除术时的平均(±标准差)年龄为3.68±2.9岁,再次(“修正”)腺样体切除术时为7.69±4.04岁,两次手术之间的平均时间间隔为4.3年。初次腺样体切除术时的年龄并非预测再次腺样体手术的重要因素。29/72(40%)接受了反流检查,包括胃排空闪烁扫描、24小时pH探头检查或喉镜检查。28/29(96%)被诊断为反流。至少15/72(21%)报告有与腺样体再生长相符的症状,发现是由咽鼓管扁桃体增生引起的。
很少需要进行再次腺样体切除术。与先前切除的残留腺样体组织再生长不同,咽鼓管扁桃体增生是部分病例的原因。食管外反流在一些病例中可能是原因之一,需要进一步研究。