Saif Imran, Halim Abdul, Altaf Ashfaq, Saif Mohsin, Siddiqui Umair, Rahman Muhammad Khalid, Azam Nadeem
Department of Nephrology, Military Hospital Rawalpindi.
J Pak Med Assoc. 2008 Jan;58(1):41-3.
A 36-year-old male developed acute renal failure and severe metabolic acidosis following acute severe gastroenteritis. Two hours following haemodialysis he developed generalized muscle weakness, which progressed to quadriplegia over the next 4 hours. Electrocardiography (ECG) revealed classic signs of hypokalaemia. Serum potassium (K+) levels were 0.98 mmol/L. He was immediately started on intravenous (i/v) and oral potassium supplementation. He gradually improved and his ECG changes also reverted as potassium levels normalized. Postdialysis hypokalaemia can be a serious complication in patients who are already in total body potassium deficit. Dialysis fluid potassium levels should be kept higher in such patients.
一名36岁男性在急性重症肠胃炎后出现急性肾衰竭和严重代谢性酸中毒。血液透析两小时后，他出现全身肌无力，并在接下来的4小时内发展为四肢瘫痪。心电图（ECG）显示出低钾血症的典型体征。血清钾（K+）水平为0.98 mmol/L。他立即开始接受静脉和口服补钾治疗。随着血钾水平恢复正常，他逐渐好转，心电图变化也恢复正常。透析后低钾血症对于已经存在全身钾缺乏的患者可能是一种严重并发症。对于这类患者，透析液中的钾水平应保持较高。
