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本文引用的文献

1
Systemic immune challenge activates an intrinsically regulated local inflammatory circuit in the adrenal gland.全身性免疫刺激激活肾上腺中一个内在调节的局部炎症回路。
Endocrinology. 2008 Apr;149(4):1436-50. doi: 10.1210/en.2007-1456. Epub 2008 Jan 3.
2
Stress induced morphological microglial activation in the rodent brain: involvement of interleukin-18.应激诱导啮齿动物大脑中的小胶质细胞形态激活：白细胞介素-18的作用。
Neuroscience. 2007 May 25;146(3):1388-99. doi: 10.1016/j.neuroscience.2007.02.043. Epub 2007 Apr 11.
3
The role of toll-like receptors in the immune-adrenal crosstalk.Toll样受体在免疫-肾上腺相互作用中的作用。
Ann N Y Acad Sci. 2006 Nov;1088:307-18. doi: 10.1196/annals.1366.027.
4
Microglia serve as a neuroimmune substrate for stress-induced potentiation of CNS pro-inflammatory cytokine responses.小胶质细胞作为应激诱导的中枢神经系统促炎细胞因子反应增强的神经免疫底物。
Brain Behav Immun. 2007 Jan;21(1):47-59. doi: 10.1016/j.bbi.2006.03.005. Epub 2006 May 2.
5
Stress-induced elevation of glucocorticoids increases microglia proliferation through NMDA receptor activation.应激诱导的糖皮质激素升高通过NMDA受体激活增加小胶质细胞增殖。
J Neuroimmunol. 2006 Feb;171(1-2):72-85. doi: 10.1016/j.jneuroim.2005.09.012. Epub 2005 Nov 8.
6
Catecholamines mediate stress-induced increases in peripheral and central inflammatory cytokines.儿茶酚胺介导应激诱导的外周和中枢炎症细胞因子增加。
Neuroscience. 2005;135(4):1295-307. doi: 10.1016/j.neuroscience.2005.06.090. Epub 2005 Sep 13.
7
Mechanisms of glucocorticoid receptor action in noninflammatory and inflammatory cells.糖皮质激素受体在非炎症细胞和炎症细胞中的作用机制。
Proc Am Thorac Soc. 2004;1(3):239-46. doi: 10.1513/pats.200402-005MS.
8
Protracted increases in core body temperature and interleukin-1 following acute administration of lipopolysaccharide: implications for the stress response.急性给予脂多糖后核心体温和白细胞介素-1的持续升高：对应激反应的影响。
Physiol Behav. 2005 Jun 30;85(3):296-307. doi: 10.1016/j.physbeh.2005.04.016.
9
Stress-induced increases in hypothalamic IL-1: a systematic analysis of multiple stressor paradigms.应激诱导的下丘脑白细胞介素-1增加：多种应激源范式的系统分析
Brain Res Bull. 2005 Jan 30;64(6):541-56. doi: 10.1016/j.brainresbull.2004.11.003.
10
Norepinephrine, the beta-adrenergic receptor, and immunity.去甲肾上腺素、β-肾上腺素能受体与免疫
Brain Behav Immun. 2002 Aug;16(4):290-332. doi: 10.1006/brbi.2001.0639.

Immune cells and cytokine circuits: toward a working model for understanding direct immune-to-adrenal communication pathways.

作者信息

Deak Terrence

机构信息

Department of Psychology, SUNY Binghamton, Vestal Parkway East, Binghamton, NY 13902-6000, USA.

出版信息

Endocrinology. 2008 Apr;149(4):1433-5. doi: 10.1210/en.2008-0170.

DOI:10.1210/en.2008-0170
PMID:18359749
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC2276715/
Abstract
摘要