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The clustering of GABA(A) receptor subtypes at inhibitory synapses is facilitated via the direct binding of receptor alpha 2 subunits to gephyrin.
J Neurosci. 2008 Feb 6;28(6):1356-65. doi: 10.1523/JNEUROSCI.5050-07.2008.
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Deficits in phosphorylation of GABA(A) receptors by intimately associated protein kinase C activity underlie compromised synaptic inhibition during status epilepticus.
J Neurosci. 2008 Jan 9;28(2):376-84. doi: 10.1523/JNEUROSCI.4346-07.2008.
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NMDA receptor activation potentiates inhibitory transmission through GABA receptor-associated protein-dependent exocytosis of GABA(A) receptors.
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GABA(A) receptor gamma 2 subunit mutations linked to human epileptic syndromes differentially affect phasic and tonic inhibition.
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Activity-dependent ubiquitination of GABA(A) receptors regulates their accumulation at synaptic sites.
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Mechanisms of reversible GABAA receptor plasticity after ethanol intoxication.
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Advances in the pathophysiology of status epilepticus.
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Impaired GABAergic transmission and altered hippocampal synaptic plasticity in collybistin-deficient mice.
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The GABAA receptor alpha1 subunit epilepsy mutation A322D inhibits transmembrane helix formation and causes proteasomal degradation.
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