CLN- and CDC28-dependent stimulation of CLN1 and CLN2 RNA levels: implications for regulation by alpha-factor and by cell cycle progression. - Suppr | 超能文献

CLN- and CDC28-dependent stimulation of CLN1 and CLN2 RNA levels: implications for regulation by alpha-factor and by cell cycle progression.

作者信息

Cross F R

机构信息

Rockefeller University, New York, New York 10021.

出版信息

Cold Spring Harb Symp Quant Biol. 1991;56:1-8. doi: 10.1101/sqb.1991.056.01.003.

DOI:10.1101/sqb.1991.056.01.003

Abstract

摘要

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Cell cycle arrest caused by CLN gene deficiency in Saccharomyces cerevisiae resembles START-I arrest and is independent of the mating-pheromone signalling pathway.酿酒酵母中CLN基因缺陷导致的细胞周期停滞类似于START-I停滞，且独立于交配信息素信号通路。

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Cdc37 is required for association of the protein kinase Cdc28 with G1 and mitotic cyclins.蛋白激酶Cdc28与G1期和有丝分裂周期蛋白的结合需要Cdc37。

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