Surgically relevant aspects of stem cell paracrine effects.
作者信息
Crisostomo Paul R, Markel Troy A, Wang Yue, Meldrum Daniel R
机构信息
Department of Surgery, Indiana University School of Medicine, Indianapolis, Ind, USA.
出版信息
Surgery. 2008 May;143(5):577-81. doi: 10.1016/j.surg.2007.10.015. Epub 2008 Jan 30.
Abstract
摘要
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In the adult mesenchymal stem cell population, source gender is a biologically relevant aspect of protective power.在成人间充质干细胞群体中，来源性别是保护能力的一个生物学相关方面。
Surgery. 2007 Aug;142(2):215-21. doi: 10.1016/j.surg.2007.04.013.
2
Activation of individual tumor necrosis factor receptors differentially affects stem cell growth factor and cytokine production.单个肿瘤坏死因子受体的激活对干细胞生长因子和细胞因子的产生有不同影响。
Am J Physiol Gastrointest Liver Physiol. 2007 Oct;293(4):G657-62. doi: 10.1152/ajpgi.00230.2007. Epub 2007 Jul 19.
3
Gender differences in injury induced mesenchymal stem cell apoptosis and VEGF, TNF, IL-6 expression: role of the 55 kDa TNF receptor (TNFR1).损伤诱导的间充质干细胞凋亡及VEGF、TNF、IL-6表达中的性别差异：55 kDa肿瘤坏死因子受体（TNFR1）的作用
J Mol Cell Cardiol. 2007 Jan;42(1):142-9. doi: 10.1016/j.yjmcc.2006.09.016. Epub 2006 Oct 30.
4
Supportive interaction between cell survival signaling and angiocompetent factors enhances donor cell survival and promotes angiomyogenesis for cardiac repair.细胞存活信号与血管生成能力因子之间的支持性相互作用可提高供体细胞的存活率，并促进血管生成性肌生成以修复心脏。
Circ Res. 2006 Sep 29;99(7):776-84. doi: 10.1161/01.RES.0000244687.97719.4f. Epub 2006 Sep 7.
5
Human progenitor cells from bone marrow or adipose tissue produce VEGF, HGF, and IGF-I in response to TNF by a p38 MAPK-dependent mechanism.来自骨髓或脂肪组织的人类祖细胞通过p38丝裂原活化蛋白激酶（p38 MAPK）依赖性机制对肿瘤坏死因子（TNF）产生血管内皮生长因子（VEGF）、肝细胞生长因子（HGF）和胰岛素样生长因子-I（IGF-I）。
Am J Physiol Regul Integr Comp Physiol. 2006 Oct;291(4):R880-4. doi: 10.1152/ajpregu.00280.2006. Epub 2006 May 25.
6
Bone marrow stem cells prevent left ventricular remodeling of ischemic heart through paracrine signaling.骨髓干细胞通过旁分泌信号传导预防缺血性心脏病的左心室重构。
Circ Res. 2006 Jun 9;98(11):1414-21. doi: 10.1161/01.RES.0000225952.61196.39. Epub 2006 May 11.
7
Pretreatment with adult progenitor cells improves recovery and decreases native myocardial proinflammatory signaling after ischemia.用成体祖细胞进行预处理可改善缺血后的恢复情况，并降低天然心肌的促炎信号传导。
Shock. 2006 May;25(5):454-9. doi: 10.1097/01.shk.0000209536.68682.90.
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Signaling factors in stem cell-mediated repair of infarcted myocardium.干细胞介导的梗死心肌修复中的信号因子
J Mol Cell Cardiol. 2005 Aug;39(2):363-76. doi: 10.1016/j.yjmcc.2005.05.012.
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Haematopoietic stem cells do not transdifferentiate into cardiac myocytes in myocardial infarcts.造血干细胞不会在心肌梗死中转化为心肌细胞。
Nature. 2004 Apr 8;428(6983):664-8. doi: 10.1038/nature02446. Epub 2004 Mar 21.
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