血小板因子 XIII 介导的纤维蛋白 - 纤维蛋白以及α2 - 抗纤溶酶 - 纤维蛋白交联增加了血小板凝块对纤维蛋白溶解的抵抗力。 - Suppr

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超能文献

Fibrin-fibrin and alpha 2-antiplasmin-fibrin cross-linking by platelet factor XIII increases the resistance of platelet clots to fibrinolysis.

作者信息

Reed G L, Matsueda G R, Haber E

机构信息

Cardiac Unit, Massachusetts General Hospital, Boston 02114.

出版信息

Trans Assoc Am Physicians. 1991;104:21-8.

PMID:1845147

Abstract

摘要

相似文献

Fibrin-fibrin and alpha 2-antiplasmin-fibrin cross-linking by platelet factor XIII increases the resistance of platelet clots to fibrinolysis.血小板因子 XIII 介导的纤维蛋白 - 纤维蛋白以及α2 - 抗纤溶酶 - 纤维蛋白交联增加了血小板凝块对纤维蛋白溶解的抵抗力。

Trans Assoc Am Physicians. 1991;104:21-8.

Platelet factor XIII increases the fibrinolytic resistance of platelet-rich clots by accelerating the crosslinking of alpha 2-antiplasmin to fibrin.血小板因子 XIII 通过加速α2-抗纤溶酶与纤维蛋白的交联来增加富含血小板凝块的纤溶抗性。

Thromb Haemost. 1992 Sep 7;68(3):315-20.

Cross-linking of alpha 2-antiplasmin to fibrin is a key factor in regulating blood clot lysis: species differences.α2-抗纤溶酶与纤维蛋白的交联是调节血凝块溶解的关键因素：种属差异。

Blood Coagul Fibrinolysis. 1993 Dec;4(6):869-75.

Release of alpha 2-plasmin inhibitor from plasma fibrin clots by activated coagulation factor XIII. Its effect on fibrinolysis.活化凝血因子XIII促使α2-纤溶酶抑制剂从血浆纤维蛋白凝块中释放。其对纤维蛋白溶解的影响。

J Clin Invest. 1986 Mar;77(3):1006-13. doi: 10.1172/JCI112352.

Cross-linking of alpha 2-plasmin inhibitor to fibrin by fibrin-stabilizing factor.纤维蛋白稳定因子使α2-纤溶酶抑制剂与纤维蛋白发生交联。

J Clin Invest. 1980 Feb;65(2):290-7. doi: 10.1172/JCI109671.

Novel aspects of platelet factor XIII function.血小板因子 XIII 功能的新方面。

Thromb Res. 2016 May;141 Suppl 2:S17-21. doi: 10.1016/S0049-3848(16)30356-5.

Thrombomodulin-dependent effect of factor V Leiden mutation on the cross-linking of α2-plasmin inhibitor to fibrin and its consequences on fibrinolysis.凝血酶调节蛋白依赖性因子 V 莱顿突变对 α2-纤溶酶抑制剂与纤维蛋白交联的影响及其对纤维蛋白溶解的后果。

Thromb Res. 2012 Sep;130(3):528-34. doi: 10.1016/j.thromres.2012.05.019. Epub 2012 Jun 15.

Lysis of clots prepared from whole blood and plasma.对由全血和血浆制备的凝块进行溶解。

Fed Proc. 1981 May 15;40(7):2092-8.

Promotion of the crosslinking of fibrin and alpha 2-antiplasmin by platelets.血小板对纤维蛋白与α2-抗纤溶酶交联的促进作用。

Thromb Haemost. 1996 Jan;75(1):161-7.

The contribution of activated factor XIII to fibrinolytic resistance in experimental pulmonary embolism.活化因子XIII在实验性肺栓塞中对纤溶抵抗的作用。

Circulation. 1999 Jan 19;99(2):299-304. doi: 10.1161/01.cir.99.2.299.

引用本文的文献

Platelet factor XIII-A regulates platelet function and promotes clot retraction and stability.血小板因子 XIII-A 调节血小板功能并促进血凝块回缩与稳定。

Res Pract Thromb Haemost. 2023 Jun 7;7(5):100200. doi: 10.1016/j.rpth.2023.100200. eCollection 2023 Jul.

All tangled up: interactions of the fibrinolytic and innate immune systems.错综复杂：纤维蛋白溶解系统与先天免疫系统的相互作用。

Front Med (Lausanne). 2023 Jun 2;10:1212201. doi: 10.3389/fmed.2023.1212201. eCollection 2023.

Persistent clotting protein pathology in Long COVID/Post-Acute Sequelae of COVID-19 (PASC) is accompanied by increased levels of antiplasmin.长期 COVID/COVID-19 后急性后遗症（PASC）中持续存在的凝血蛋白病理学伴随着抗纤溶酶水平的升高。

Cardiovasc Diabetol. 2021 Aug 23;20(1):172. doi: 10.1186/s12933-021-01359-7.

Factor XIII-A: An Indispensable "Factor" in Haemostasis and Wound Healing.凝血因子 XIII-A：止血和伤口愈合中不可或缺的“因子”

Int J Mol Sci. 2021 Mar 17;22(6):3055. doi: 10.3390/ijms22063055.

Alpha2-Antiplasmin: The Devil You Don't Know in Cerebrovascular and Cardiovascular Disease.α2-抗纤溶酶：脑血管病和心血管病中你所不了解的“恶魔”

Front Cardiovasc Med. 2020 Dec 23;7:608899. doi: 10.3389/fcvm.2020.608899. eCollection 2020.

Factor XIII deficiency does not prevent FeCl-induced carotid artery thrombus formation in mice.因子 XIII 缺乏并不妨碍小鼠体内由 FeCl 诱导的颈动脉血栓形成。

Res Pract Thromb Haemost. 2019 Nov 6;4(1):111-116. doi: 10.1002/rth2.12278. eCollection 2020 Jan.

Factor XIII in plasma, but not in platelets, mediates red blood cell retention in clots and venous thrombus size in mice.血浆中的因子 XIII，而不是血小板，介导了红细胞在血栓中的滞留和静脉血栓的大小。

Blood Adv. 2018 Jan 3;2(1):25-35. doi: 10.1182/bloodadvances.2017011890. eCollection 2018 Jan 9.

Functional factor XIII-A is exposed on the stimulated platelet surface.功能性凝血因子ⅩⅢ -A暴露于受刺激的血小板表面。

Blood. 2014 Dec 18;124(26):3982-90. doi: 10.1182/blood-2014-06-583070. Epub 2014 Oct 20.

Microvascular thrombosis, fibrinolysis, ischemic injury, and death after cerebral thromboembolism are affected by levels of circulating α2-antiplasmin.微血管血栓形成、纤维蛋白溶解、缺血性损伤以及脑栓子形成后的死亡都受到循环中α2-抗纤溶酶水平的影响。

Arterioscler Thromb Vasc Biol. 2014 Dec;34(12):2586-93. doi: 10.1161/ATVBAHA.114.304530. Epub 2014 Sep 25.

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