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溶酶体肌病:自噬体中过度堆积的物质难以处理。

Lysosomal myopathies: an excessive build-up in autophagosomes is too much to handle.

作者信息

Malicdan May Christine, Noguchi Satoru, Nonaka Ikuya, Saftig Paul, Nishino Ichizo

机构信息

Department of Neuromuscular Research, National Institute of Neuroscience, National Center of Neurology and Psychiatry, Kodaira, Tokyo 187-8502, Japan.

出版信息

Neuromuscul Disord. 2008 Jul;18(7):521-9. doi: 10.1016/j.nmd.2008.04.010. Epub 2008 May 27.

DOI:10.1016/j.nmd.2008.04.010
PMID:18502640
Abstract

Lysosomes are membrane-bound acidic organelles that contain hydrolases used for intracellular digestion of various macromolecules in a process generally referred to as autophagy. In normal skeletal and cardiac muscles, lysosomes usually appear morphologically unremarkable and thus are not readily visible on light microscopy. In distinct neuromuscular disorders, however, lysosomes have been shown to be structurally abnormal and functionally impaired, leading to the accumulation of autophagic vacuoles in myofibers. More specifically, there are myopathies in which buildup of these autophagic vacuoles seem to predominate the pathological picture. In such conditions, autophagy is considered not merely a secondary event, but a phenomenon that actually contributes to disease pathomechanism and/or progression. At present, there are two disorders in the muscle which are associated with primary defect in lysosomal proteins, namely Danon disease and Pompe disease. Other myopathies which have prominent autophagy in the skeletal muscle include X-linked myopathy with excessive autophagy (XMEA). In this review, these disorders are briefly characterized, and the role of autophagy in the context of the pathomechanism of these disorders is highlighted.

摘要

溶酶体是膜结合的酸性细胞器,含有水解酶,用于在通常称为自噬的过程中对各种大分子进行细胞内消化。在正常的骨骼肌和心肌中,溶酶体通常在形态上无明显异常,因此在光学显微镜下不易看到。然而,在某些特定的神经肌肉疾病中,溶酶体已被证明存在结构异常和功能受损,导致自噬泡在肌纤维中积累。更具体地说,有一些肌病,其中这些自噬泡的积累似乎在病理图像中占主导地位。在这种情况下,自噬不仅被认为是一个次要事件,而且是一种实际上促成疾病发病机制和/或进展的现象。目前,肌肉中有两种疾病与溶酶体蛋白的原发性缺陷有关,即丹侬病和庞贝病。骨骼肌中具有显著自噬的其他肌病包括X连锁过度自噬性肌病(XMEA)。在这篇综述中,简要描述了这些疾病的特征,并强调了自噬在这些疾病发病机制中的作用。

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