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Malignant transformation of human keratinocytes during adaptation to autotrophy.

作者信息

Hill M, Hillova J, Mariage-Samson R

出版信息

In Vitro Cell Dev Biol. 1991 Apr;27A(4):270-2. doi: 10.1007/BF02630901.

DOI:10.1007/BF02630901
PMID:1856151
Abstract
摘要

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1
Malignant transformation of human keratinocytes during adaptation to autotrophy.人类角质形成细胞在适应自养过程中的恶性转化。
In Vitro Cell Dev Biol. 1991 Apr;27A(4):270-2. doi: 10.1007/BF02630901.
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Growth in agar and tumor formation in immunologically incompetent mice as criteria for keratinocyte transformation.以免疫缺陷小鼠中琼脂生长和肿瘤形成作为角质形成细胞转化的标准。
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Smad7 but not Smad6 cooperates with oncogenic ras to cause malignant conversion in a mouse model for squamous cell carcinoma.在鳞状细胞癌小鼠模型中，Smad7而非Smad6与致癌性Ras协同作用导致恶性转化。
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Malignant transformation of immortalized HaCaT keratinocytes through deregulated nuclear factor kappaB signaling.通过失调的核因子κB信号通路使永生化的HaCaT角质形成细胞发生恶性转化。
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Stage-specific effect of N-(4-hydroxyphenyl)retinamide on cell growth in squamous cell carcinogenesis.N-(4-羟基苯基)视黄酸对鳞状细胞癌发生过程中细胞生长的阶段特异性作用。
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Strain-dependent differences in malignant conversion of mouse skin tumors is an inherent property of the epidermal keratinocyte.小鼠皮肤肿瘤恶性转化中应变依赖性差异是表皮角质形成细胞的固有特性。
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Two oncogenes, v-fos and v-ras, cooperate to convert normal keratinocytes to squamous cell carcinoma.两种癌基因，v-fos和v-ras，协同作用将正常角质形成细胞转化为鳞状细胞癌。
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引用本文的文献

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Tumor progression of skin carcinoma cells in vivo promoted by clonal selection, mutagenesis, and autocrine growth regulation by granulocyte colony-stimulating factor and granulocyte-macrophage colony-stimulating factor.克隆选择、诱变以及粒细胞集落刺激因子和粒细胞-巨噬细胞集落刺激因子的自分泌生长调节促进皮肤癌细胞在体内的肿瘤进展。
Am J Pathol. 2001 Oct;159(4):1567-79. doi: 10.1016/S0002-9440(10)62541-2.
2
A free-radical hypothesis for the instability and evolution of genotype and phenotype in vitro.体外基因型和表型不稳定性及进化的自由基假说
Cytotechnology. 1992;10(2):93-124. doi: 10.1007/BF00570888.

本文引用的文献

1
Epithelial line from normal human colon mucosa.来自正常人类结肠黏膜的上皮细胞系。
J Natl Cancer Inst. 1982 Dec;69(6):1271-6.
2
Molecular and cytogenetic analysis of immortalized human primary keratinocytes obtained after transfection with human papillomavirus type 16 DNA.用16型人乳头瘤病毒DNA转染后获得的永生化人原代表皮细胞的分子和细胞遗传学分析
Oncogene. 1987;1(3):251-6.
3
Neoplastic transformation of human epidermal keratinocytes by AD12-SV40 and Kirsten sarcoma viruses.
Science. 1985 Mar 8;227(4691):1250-2. doi: 10.1126/science.2579430.
4
Glucocorticoid-enhanced neoplastic transformation of human keratinocytes by human papillomavirus type 16 and an activated ras oncogene.
Virology. 1989 Dec;173(2):767-71. doi: 10.1016/0042-6822(89)90595-3.
5
Induction of human cervical squamous cell carcinoma by sequential transfection with human papillomavirus 16 DNA and viral Harvey ras.通过人乳头瘤病毒16型DNA和病毒哈维鼠肉瘤病毒癌基因（v-Ha-ras）的连续转染诱导人宫颈鳞状细胞癌
Oncogene. 1989 Apr;4(4):395-9.
6
Density-dependent modulation of synthesis of keratins 1 and 10 in the human keratinocyte line HACAT and in ras-transfected tumorigenic clones.人角质形成细胞系HACAT和ras转染的致瘤克隆中角蛋白1和角蛋白10合成的密度依赖性调节
Differentiation. 1989 Mar;40(1):42-54. doi: 10.1111/j.1432-0436.1989.tb00812.x.
7
Differential effects of human papillomavirus type 6, 16, and 18 DNAs on immortalization and transformation of human cervical epithelial cells.人乳头瘤病毒6型、16型和18型DNA对人宫颈上皮细胞永生化和转化的差异影响。
Proc Natl Acad Sci U S A. 1989 Jan;86(2):563-7. doi: 10.1073/pnas.86.2.563.
8
Continuous cell lines with altered growth and differentiation properties originate after transfection of human keratinocytes with human papillomavirus type 16 DNA.具有改变的生长和分化特性的连续细胞系起源于人乳头瘤病毒16型DNA转染人角质形成细胞之后。
Carcinogenesis. 1988 Sep;9(9):1573-9. doi: 10.1093/carcin/9.9.1573.
9
Characterization of normal human exocervical epithelial cells immortalized in vitro by papillomavirus types 16 and 18 DNA.人乳头瘤病毒16型和18型DNA体外永生化的正常人外宫颈上皮细胞的特性分析
Cancer Res. 1988 Aug 15;48(16):4620-8.
10
Characterization of primary human keratinocytes transformed by human papillomavirus type 18.人乳头瘤病毒18型转化的原代人角质形成细胞的特性分析
J Virol. 1988 Jun;62(6):1917-24. doi: 10.1128/JVI.62.6.1917-1924.1988.