相似文献
1
Optical induction of plasticity at single synapses reveals input-specific accumulation of alphaCaMKII.
Proc Natl Acad Sci U S A. 2008 Aug 19;105(33):12039-44. doi: 10.1073/pnas.0802940105. Epub 2008 Aug 12.
2
Activation of CaMKII in single dendritic spines during long-term potentiation.
Nature. 2009 Mar 19;458(7236):299-304. doi: 10.1038/nature07842.
3
βCaMKII plays a nonenzymatic role in hippocampal synaptic plasticity and learning by targeting αCaMKII to synapses.
J Neurosci. 2011 Jul 13;31(28):10141-8. doi: 10.1523/JNEUROSCI.5105-10.2011.
4
Long-term Memory Upscales Volume of Postsynaptic Densities in the Process that Requires Autophosphorylation of αCaMKII.
Cereb Cortex. 2020 Apr 14;30(4):2573-2585. doi: 10.1093/cercor/bhz261.
5
Synaptic strength of individual spines correlates with bound Ca2+-calmodulin-dependent kinase II.
J Neurosci. 2007 Dec 19;27(51):14007-11. doi: 10.1523/JNEUROSCI.3587-07.2007.
6
Local, persistent activation of Rho GTPases during plasticity of single dendritic spines.
Nature. 2011 Apr 7;472(7341):100-4. doi: 10.1038/nature09823. Epub 2011 Mar 20.
7
Autocrine BDNF-TrkB signalling within a single dendritic spine.
Nature. 2016 Oct 6;538(7623):99-103. doi: 10.1038/nature19766. Epub 2016 Sep 28.
8
Persistent accumulation of calcium/calmodulin-dependent protein kinase II in dendritic spines after induction of NMDA receptor-dependent chemical long-term potentiation.
J Neurosci. 2004 Oct 20;24(42):9324-31. doi: 10.1523/JNEUROSCI.2350-04.2004.
9
Protein synthesis and neurotrophin-dependent structural plasticity of single dendritic spines.
Science. 2008 Mar 21;319(5870):1683-7. doi: 10.1126/science.1152864. Epub 2008 Feb 28.
10
Role of inhibitory autophosphorylation of calcium/calmodulin-dependent kinase II (αCAMKII) in persistent (>24 h) hippocampal LTP and in LTD facilitated by novel object-place learning and recognition in mice.
Behav Brain Res. 2015 May 15;285:79-88. doi: 10.1016/j.bbr.2014.01.022. Epub 2014 Jan 27.
引用本文的文献
1
Micro-sub regional synapse weakening by mimicking the hyperphosphorylation of microtubule associated protein Tau in dendritic spines.
Brain Commun. 2025 Jun 11;7(3):fcaf234. doi: 10.1093/braincomms/fcaf234. eCollection 2025.
2
Split genetically encoded calcium indicators for interorganellar junctions.
Proc Natl Acad Sci U S A. 2025 May 20;122(20):e2415268122. doi: 10.1073/pnas.2415268122. Epub 2025 May 13.
3
Targeting Tiam1 Enhances Hippocampal-Dependent Learning and Memory in the Adult Brain and Promotes NMDA Receptor-Mediated Synaptic Plasticity and Function.
J Neurosci. 2025 Feb 5;45(6):e0298242024. doi: 10.1523/JNEUROSCI.0298-24.2024.
4
LTP expression mediated by autonomous activity of GluN2B-bound CaMKII.
Cell Rep. 2024 Oct 22;43(10):114866. doi: 10.1016/j.celrep.2024.114866. Epub 2024 Oct 11.
5
LOV2-based photoactivatable CaMKII and its application to single synapses: Local Optogenetics.
Biophys Physicobiol. 2023 Jun 6;20(2):e200027. doi: 10.2142/biophysico.bppb-v20.0027. eCollection 2023.
6
Synaptic memory and CaMKII.
Physiol Rev. 2023 Oct 1;103(4):2877-2925. doi: 10.1152/physrev.00034.2022. Epub 2023 Jun 8.
7
Activation of Ventral Pallidum CaMKIIa-Expressing Neurons Promotes Wakefulness.
Neurochem Res. 2023 Aug;48(8):2502-2513. doi: 10.1007/s11064-023-03915-x. Epub 2023 Apr 5.
8
Wireless multi-lateral optofluidic microsystems for real-time programmable optogenetics and photopharmacology.
Nat Commun. 2022 Sep 22;13(1):5571. doi: 10.1038/s41467-022-32947-0.
9
Förster resonance energy transfer-based kinase mutation phenotyping reveals an aberrant facilitation of Ca/calmodulin-dependent CaMKIIα activity in mutations related to intellectual disability.
Front Mol Neurosci. 2022 Sep 1;15:970031. doi: 10.3389/fnmol.2022.970031. eCollection 2022.
10
CaMKII: a central molecular organizer of synaptic plasticity, learning and memory.
Nat Rev Neurosci. 2022 Nov;23(11):666-682. doi: 10.1038/s41583-022-00624-2. Epub 2022 Sep 2.
本文引用的文献
1
Locally dynamic synaptic learning rules in pyramidal neuron dendrites.
Nature. 2007 Dec 20;450(7173):1195-200. doi: 10.1038/nature06416.
2
Synaptic strength of individual spines correlates with bound Ca2+-calmodulin-dependent kinase II.
J Neurosci. 2007 Dec 19;27(51):14007-11. doi: 10.1523/JNEUROSCI.3587-07.2007.
3
A structural mechanism for maintaining the 'on-state' of the CaMKII memory switch in the post-synaptic density.
J Neurochem. 2007 Oct;103(1):357-64. doi: 10.1111/j.1471-4159.2007.04744.x.
4
Reversal of synaptic memory by Ca2+/calmodulin-dependent protein kinase II inhibitor.
J Neurosci. 2007 May 9;27(19):5190-9. doi: 10.1523/JNEUROSCI.5049-06.2007.
5
Optical induction of synaptic plasticity using a light-sensitive channel.
Nat Methods. 2007 Feb;4(2):139-41. doi: 10.1038/nmeth988. Epub 2006 Dec 31.
6
CaMKII translocation requires local NMDA receptor-mediated Ca2+ signaling.
EMBO J. 2006 Dec 13;25(24):5873-83. doi: 10.1038/sj.emboj.7601420. Epub 2006 Nov 23.
7
Postsynaptic protein mobility in dendritic spines: long-term regulation by synaptic NMDA receptor activation.
Mol Cell Neurosci. 2006 Apr;31(4):702-12. doi: 10.1016/j.mcn.2006.01.010. Epub 2006 Feb 28.
8
Glutamate receptor exocytosis and spine enlargement during chemically induced long-term potentiation.
J Neurosci. 2006 Feb 15;26(7):2000-9. doi: 10.1523/JNEUROSCI.3918-05.2006.
9
Transition from reversible to persistent binding of CaMKII to postsynaptic sites and NR2B.
J Neurosci. 2006 Jan 25;26(4):1164-74. doi: 10.1523/JNEUROSCI.3116-05.2006.
10
Neuronal activity regulates diffusion across the neck of dendritic spines.
Science. 2005 Nov 4;310(5749):866-9. doi: 10.1126/science.1114816.
Zotero 插件