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缩窄性心包炎压迫并使升主动脉变窄。

Constrictive pericarditis impressing and narrowing the ascending aorta.

作者信息

Tugcu Aylin, Yildirimturk Ozlem, Duran Cihan, Aytekin Saide

机构信息

Department of Cardiology, Florence Nightingale Hospital, Istanbul, Turkey.

出版信息

Echocardiography. 2008 Aug;25(7):768-71. doi: 10.1111/j.1540-8175.2008.00660.x.

Abstract

A 77-year-old male patient was admitted to our institution with 1-year history of progressive dyspnea on exertion, and lower extremity edema. His chest x-ray showed a circumferential pericardial calcification and right-sided pleural effusion. The electrocardiography revealed atrial fibrillation with low voltage in all derivations and diffuse nonspecific T-wave inversions. The transesophageal echocardiography showed a thickened pericardium with biatrial enlargement and normal right and left ventricular systolic functions. A thick echogenic structure that caused impression and narrowing of the ascending aorta was observed. Simultaneous right and left heart catheterization showed elevation and equalization of right-sided and left-sided diastolic filling pressures, with characteristic dip and plateau. Aortic angiogram showed the ascending aorta was impressed and narrowed by calcified pericardium. Cine magnetic resonance imaging showed pericardial calcifications impressing and narrowing of the ascending aorta. All these findings were consistent with constrictive pericarditis. The patient had no history of tuberculosis, cardiac surgery, or mediastinal irradiation. His HIV antibody test was negative. Marked pericardial thickening and calcifications were evident during pericardiectomy. Histological analysis of the pericardium showed dense collageneous matrix, mild chronic inflammation and calcification. The culture of pericardial tissue revealed no identifiable cause including tuberculosis. The patient was diagnosed as idiopathic constrictive pericarditis. The patient's symptoms and edema decreased remarkably after pericardial stripping. He remained well at 1-year follow-up.

摘要

一名77岁男性患者因进行性劳力性呼吸困难和下肢水肿1年入住我院。胸部X线显示心包环形钙化及右侧胸腔积液。心电图显示房颤,各导联低电压,弥漫性非特异性T波倒置。经食管超声心动图显示心包增厚,双房扩大,右、左心室收缩功能正常。观察到一个增厚的强回声结构压迫并使升主动脉变窄。同时进行的右心和左心导管检查显示右侧和左侧舒张期充盈压升高且相等,呈典型的下陷和高原波形。主动脉血管造影显示升主动脉被钙化的心包压迫并变窄。电影磁共振成像显示心包钙化压迫并使升主动脉变窄。所有这些发现均符合缩窄性心包炎。该患者无结核、心脏手术或纵隔放疗史。其HIV抗体检测为阴性。心包切除术中可见明显的心包增厚和钙化。心包组织学分析显示致密的胶原基质、轻度慢性炎症和钙化。心包组织培养未发现包括结核在内的可识别病因。该患者被诊断为特发性缩窄性心包炎。心包剥脱术后患者的症状和水肿明显减轻。1年随访时患者情况良好。

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