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[自由基在维生素D的转化及生理作用机制中的可能作用]

[Possible role of free radicals in the transformations and mechanism of physiological action of vitamin D].

作者信息

Spirichev V B, Gazdarov A K, Saprin A N, Blazheevich N V

出版信息

Biofizika. 1976 Jul-Aug;21(4):676-8.

PMID:188487
Abstract

Deficiency of vitamine D is accompanied with a decrease of ESR signal intensity from g = 2.003, as well as of the signals with g = 1.94 and gav. = 2.25 in the liver and kidneys of young rats. Injection of 2.5 g ergocalciferole (vitamine D2) to these animals results in a complete or partial normalization of the intensity of these signals during 6--16 hrs and an increase of the ESR signal intensity from g = 2.003 in small intestine mucosa.

摘要

维生素D缺乏伴随着幼鼠肝脏和肾脏中g = 2.003的电子自旋共振(ESR)信号强度以及g = 1.94和gav. = 2.25信号强度的降低。给这些动物注射2.5克麦角钙化醇(维生素D2)后,这些信号强度在6至16小时内完全或部分恢复正常,并且小肠黏膜中g = 2.003的ESR信号强度增加。

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