• 文献检索
  • 文档翻译
  • 深度研究
  • 学术资讯
  • Suppr Zotero 插件Zotero 插件
  • 邀请有礼
  • 套餐&价格
  • 历史记录
应用&插件
Suppr Zotero 插件Zotero 插件浏览器插件Mac 客户端Windows 客户端微信小程序
定价
高级版会员购买积分包购买API积分包
服务
文献检索文档翻译深度研究API 文档MCP 服务
关于我们
关于 Suppr公司介绍联系我们用户协议隐私条款
关注我们

Suppr 超能文献

核心技术专利：CN118964589B侵权必究
粤ICP备2023148730 号-1Suppr @ 2026

文献检索

告别复杂PubMed语法，用中文像聊天一样搜索，搜遍4000万医学文献。AI智能推荐，让科研检索更轻松。

立即免费搜索

文件翻译

保留排版，准确专业，支持PDF/Word/PPT等文件格式，支持 12+语言互译。

免费翻译文档

深度研究

AI帮你快速写综述，25分钟生成高质量综述，智能提取关键信息，辅助科研写作。

立即免费体验

[适应周期性低氧对心肌梗死后血压下降及内皮细胞过度激活的影响]

[Effect of adaptation to periodic hypoxia on post-infarction fall of blood pressure and hyperactivation of the endothelium].

作者信息

Manukhina E B, Lapshin A V, Meerson F Z

出版信息

Fiziol Zh (1978). 1991 May-Jun;37(3):98-105.

PMID:1894058
Abstract

Acute stress concomitant to the experimental myocardial infarction has induced endothelial hyperactivation of the rat aorta exhibited in an increase of inhibition of norepinephrine-induced contractions of vascular smooth muscle, enhanced endothelium-dependent relaxation correlating with a fall of systemic blood pressure. Preliminary adaptation of rats to intermittent hypobaric hypoxia greatly prevented the stress-induced endothelial hyperactivation and beneficially affected the postinfarction time course of blood pressure.

摘要

实验性心肌梗死伴随的急性应激已诱导大鼠主动脉内皮细胞过度激活，表现为去甲肾上腺素诱导的血管平滑肌收缩抑制增加、内皮依赖性舒张增强，这与全身血压下降相关。大鼠对间歇性低压缺氧的初步适应极大地预防了应激诱导的内皮细胞过度激活，并对心肌梗死后的血压变化过程产生有益影响。

相似文献

1
[Effect of adaptation to periodic hypoxia on post-infarction fall of blood pressure and hyperactivation of the endothelium].[适应周期性低氧对心肌梗死后血压下降及内皮细胞过度激活的影响]
Fiziol Zh (1978). 1991 May-Jun;37(3):98-105.
2
[Adaptation to short stress exposures prevents the enhancement of the endothelium-dependent reactions of the aorta in myocardial infarct].[适应短期应激暴露可预防心肌梗死时主动脉内皮依赖性反应的增强]
Fiziol Zh SSSR Im I M Sechenova. 1991 Mar;77(3):70-8.
3
[The prevention of a postinfarct fall in pressure and hyperactivation of the aortic endothelium in the rat by using the antioxidant ionol].[使用抗氧化剂维生素E防止大鼠心肌梗死后血压下降和主动脉内皮过度激活]
Fiziol Zh SSSR Im I M Sechenova. 1991 Jul;77(7):48-56.
4
[The limitation of acute hypotension and hyperactivation of the vascular endothelium in rats with an experimental myocardial infarct by using preliminary adaptation to physical load].[通过对体力负荷进行初步适应性训练来限制实验性心肌梗死大鼠的急性低血压和血管内皮过度激活]
Fiziol Zh Im I M Sechenova. 1996 Dec;82(12):57-64.
5
Adaptation to short-term stress prevents post-infarction hyperactivation of the endothelium and decrease in blood pressure in rats.适应短期应激可预防大鼠心肌梗死后内皮细胞过度激活及血压降低。
Biomed Sci. 1991;2(6):623-8.
6
[Correction of NO-dependent cardiovascular disorders by adaptation to hypoxia].[通过适应低氧纠正一氧化氮依赖性心血管疾病]
Ross Fiziol Zh Im I M Sechenova. 2001 Jan;87(1):110-7.
7
Role of nitric oxide in adaptation to hypoxia and adaptive defense.一氧化氮在低氧适应及适应性防御中的作用。
Physiol Res. 2000;49(1):89-97.
8
[Correlations between the endothelium-dependent relaxation of the aorta and arterial pressure in rats with myocardial infarction].[心肌梗死大鼠主动脉内皮依赖性舒张与动脉血压之间的相关性]
Biull Eksp Biol Med. 1989 Jul;108(7):21-4.
9
[The effect of experimental infarct and emotional-pain stress on the endothelium-dependent reactions of the isolated rat aorta].[实验性梗死和情绪性疼痛应激对离体大鼠主动脉内皮依赖性反应的影响]
Fiziol Zh SSSR Im I M Sechenova. 1989 Oct;75(10):1409-16.
10
[Decrease in constrictor response and increase in dilator response of the resistant artery in experimental myocardial infarction: effect of adaptation to hypoxia on this phenomenon].[实验性心肌梗死时阻力动脉收缩反应降低及舒张反应增强：缺氧适应对该现象的影响]
Biull Eksp Biol Med. 1993 May;115(5):464-6.

引用本文的文献

1
Chronic stress improves NO- and Ca2+ flux-dependent vascular function: a pharmacological study.慢性应激改善一氧化氮和钙离子流依赖性血管功能：一项药理学研究。
Arq Bras Cardiol. 2015 Mar;104(3):226-33. doi: 10.5935/abc.20140207. Epub 2015 Jan 23.