Hicks R J, Young W, Shapiro B, Kuhl D E
Department of Internal Medicine (Division of Nuclear Medicine), University of Michigan Medical Center, Ann Arbor 48109-0028.
J Nucl Med. 1991 Mar;32(3):524-6.
An incidental finding of absent splenic uptake of autologous, indium-111-oxine-labeled leukocytes in an immunosuppressed renal transplant recipient was documented to be associated with functional asplenia based on absence of technetium-99m-sulfur colloid clearance by a morphologically normal spleen. The patient had recently suffered an episode of disseminated varicella infection that might have led to the development of functional asplenia.
在一名免疫抑制的肾移植受者中，偶然发现自体铟 - 111 - 奥克辛标记的白细胞在脾脏摄取缺失，基于形态正常的脾脏对锝 - 99m - 硫胶体清除功能缺失，该发现被证明与功能性无脾相关。该患者近期曾发生播散性水痘感染，这可能导致了功能性无脾的发生。
